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Research 30 of 393
- Localization of mechanosensitive channel TRPV4 in mouse skin
- TRPV3 as a Therapeutic Target for Itch
- Activation of TRPV3 Regulates Inflammatory Actions of Human Epidermal Keratinocytes
- Influence of TRPV3 mutation on hair growth cycle in mice
- Structural mechanism of TRPV3 channel inhibition by the plant‐derived coumarin osthole
- Activation of TRPV3 Inhibits Lipogenesis and Stimulates Production of Inflammatory Mediators in Human Sebocytes—A Putative Contributor to Dry Skin Dermatoses
- TRPV4 Is Expressed in Human Hair Follicles and Inhibits Hair Growth In Vitro
- Molecular determinants for the chemical activation of the warmth-sensitive TRPV3 channel by the natural monoterpenoid carvacrol
- Mechanisms of proton inhibition and sensitization of the cation channel TRPV3
- TRPV Channels in Osteoarthritis: A Comprehensive Review
- Unlocking the therapeutic potential of TRPV3: Insights into thermosensation, channel modulation, and skin homeostasis involving TRPV3
- Different Ligands of the TRPV3 Cation Channel Cause Distinct Conformational Changes as Revealed by Intrinsic Tryptophan Fluorescence Quenching
- Transient stimulation of TRPV4‐expressing keratinocytes promotes hair follicle regeneration in mice
- Therapeutic inhibition of keratinocyte TRPV3 sensory channel by local anesthetic dyclonine
- TRPV3 Ion Channel: From Gene to Pharmacology
- The Multifaceted Functions of TRPV4 and Calcium Oscillations in Tissue Repair
- Cutaneous TRPV4 Potentiates the Afferent Transmission of Electroacupuncture Stimulation
- Pathogenesis and management of TRPV3-related Olmsted syndrome
- Whole genome sequencing analysis of alpaca suggests TRPV3 as a candidate gene for the suri phenotype
- Molecular studies of transient receptor potential Vanilloid 3 (TRPV3)
- Nociceptive and Nonnociceptive Roles of TRPV3 and Its “Druggability”
- TRPV1 Function in Health and Disease
- Olmsted Syndrome Caused by a Homozygous Recessive Mutation in TRPV3
- Hair Cycle Control by Vanilloid Receptor-1 (TRPV1): Evidence from TRPV1 Knockout Mice
- Hair Loss Caused by Gain-of-Function Mutant TRPV3 Is Associated with Premature Differentiation of Follicular Keratinocytes
- Dermal TRPV1 innervations engage a macrophage- and fibroblast-containing pathway to activate hair growth in mice
- A combination of low-dose systemic etretinate and topical calcipotriol/betamethasone dipropionate treatment for hyperkeratosis and itching in Olmsted syndrome associated with a TRPV3 mutation
- Natural phenylethanoid glycoside forsythoside A alleviates androgenetic alopecia by selectively inhibiting TRPV3 channels in mice
- Case report: Novel p.Val306Met missense mutation in TRPV3 in a case of Olmsted syndrome accompanied by squamous cell carcinoma
- 738 Cryo-EM and molecular dynamics of TRPV3 channel in activated state reveal structural features implicated in severe itch and hyperkeratosis