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ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

Indian Hedgehog helps control skin cell growth and protects against aggressive skin cancer.

Removing integrin α3β1 from hair stem cells lowers skin tumor growth by affecting CCN2 protein levels.

TAF4 is important for skin cell growth and helps prevent skin cancer in mice.

Meis1 is crucial for skin health and tumor development.

The risk of skin tumors becoming malignant depends on the specific skin cell type affected.

Mice with human skin protein K8 had more skin problems and cancer.

Blocking COX, especially COX-2, in the skin can reduce inflammation and pain and may help prevent skin cancer.

Active oxygen species might be involved in skin tumor growth, but their exact role is unclear.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

SOX2 is crucial for skin cell function and hair growth, and it plays a role in skin cancer and wound healing.

Inhibiting ODC can prevent UV-induced skin cancer.

Increased ODC activity leads to skin tumors by recruiting stem cells, not by toxic byproducts.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Retinoic acid-binding proteins in skin are regulated by β-catenin and Notch signalling.

Overexpressing ornithine decarboxylase and v-Ha-ras in keratinocytes leads to invasiveness and malignancy.

Oncogenic K-ras causes rapid cancerous changes in the mouth's lining.

Disrupting Stat3 in hair follicle stem cells greatly reduces skin tumor formation.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Understanding where cancer cells come from helps create better prevention and treatment methods.

The skin protects the body and is constantly renewed by stem cells; disruptions can lead to cancer.

Green tea may help with skin health and protect against UV damage, but more research is needed to confirm its safety and effectiveness.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Suppressing very long chain fatty acids is linked to skin cancer.

The oncoprotein causes abnormal hair growth without increasing skin cancer risk.

EGF controls hair growth by regulating hair follicles' growth phases.

Lacking cyclin D3 reduces skin cancer growth without affecting normal skin cell growth.

Inactive hair follicle stem cells help prevent skin cancer.

Mutant cells in hair follicles are influenced by their location and interactions with surrounding cells.