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LED light therapy at 863 nm wavelength can slow down skin tumor growth and reduce inflammation in mice.

Disrupting Stat3 in hair follicle stem cells greatly reduces skin tumor formation.

Lacking cyclin D3 reduces skin cancer growth without affecting normal skin cell growth.

High levels of ornithine decarboxylase can cause tumors in mouse skin.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Removing integrin α3β1 from hair stem cells lowers skin tumor growth by affecting CCN2 protein levels.

Activin increases skin tumor formation, skin Tregs help hair growth, lymph-node removal doesn't improve melanoma survival, cells can revert to stem cells in wound healing, and skin bacteria produce peptides that may treat infections.

Autophagy in skin cells is important for preventing inflammation, skin tumors, and controlling hair growth timing.

Autophagy helps control skin inflammation and cancer responses and regulates hair growth by affecting stem cell activity.

Indian Hedgehog helps control skin cell growth and protects against aggressive skin cancer.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

UV exposure reduces Lgr6+ stem cells in mouse skin and they don't significantly contribute to skin cancer development.

Inactive hair follicle stem cells help prevent skin cancer.

Inhibiting ODC can prevent UV-induced skin cancer.

The document concludes that identifying the specific cells where skin cancers begin is important for creating better prevention, detection, and treatment methods.

YAP and TAZ proteins are necessary for the development of two types of skin cancer.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive forestomach cancer.

Patched1 helps prevent tumors by controlling cell growth.

Active oxygen species might be involved in skin tumor growth, but their exact role is unclear.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Different types of skin cells have unique genetic markers that affect how likely they are to spread cancer.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

TPA is about 50 times more effective at promoting tumors than MZ.

Vitamin D receptor helps control hair growth and could be used to treat certain skin tumors.

The skin protects the body and is constantly renewed by stem cells; disruptions can lead to cancer.

The Wnt signaling pathway is not a major factor in the development of keratoacanthoma, a type of skin tumor.