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Rats can't grow new hair follicles after skin wounds, unlike mice, due to differences in gene expression and response to WNT signaling.

The African spiny mouse can fully regenerate its muscle without scarring, unlike the common house mouse.

Hair can regrow in large wounds through a process similar to how hair forms in embryos, and understanding this could lead to new treatments for hair loss or scarring.

Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

The research found that different types of fibroblasts are involved in wound healing and that some blood cells can turn into fat cells during this process.

Fibroblast Growth Factors (FGFs) are important for tissue repair and regeneration, influencing cell behavior and other factors involved in healing, and are crucial in processes like wound healing, bone repair, and hair growth.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

Mice genetically modified to produce more Del1 protein had faster hair regrowth.

Prostaglandin D2 blocks new hair growth after skin injury through the Gpr44 receptor.

Skin fat has important roles in hair growth, skin repair, immune defense, and aging, and could be targeted for skin and hair treatments.

Mice can grow new hair follicles after skin wounds through a process not involving existing hair stem cells, but requiring more research to understand fully.

Applying calreticulin can speed up wound healing in diabetics.

CD4+ skin cells may be precursors to basal cell carcinoma.

Skin and hair renewal is maintained by both fast and slow cycling stem cells, with hair regrowth primarily driven by specific stem cells in the hair follicle bulge. These cells can also help heal wounds and potentially treat hair loss.

Integrin alphavbeta6 is important for wound healing and hair growth, and blocking it may improve these processes.

The document concludes that the understanding of scar formation is incomplete and current prevention and treatment for hypertrophic scars and keloids are not fully effective.

Some medicinal plants can help heal wounds and may lead to new treatments.

Regulatory T cells are essential for normal and improved wound healing in mice.

The spiny mouse can regenerate its skin without scarring, which could help us learn how to heal human skin better.

Sebaceous glands can heal and regenerate after injury using their own stem cells and help from hair follicle cells.

Adipocytes can change into fibroblast-like cells to help with wound healing.

Silk sericin dressing with collagen heals wounds faster and improves scar quality better than Bactigras.

Verteporfin treatment in mice led to complete skin healing without scarring.

The right cells and signals can potentially lead to scarless wound healing, with a mix of natural and external wound healing controllers possibly being the best way to achieve this.

Hydrogel scaffolds can help wounds heal better and grow hair.

Understanding how baby skin heals without scars could help develop treatments for adults to heal wounds without leaving scars.

Activating Nrf2 in skin cells speeds up wound healing by increasing the growth of certain stem cells.

Four specific genes are linked to keloid formation and could be potential treatment targets.

Self-assembling RADA16-I hydrogels with bioactive peptides significantly improve wound healing.

Smad2/3-dependent TGF-β signaling increases during wound healing.