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Radiation treatment causes skin fibrosis by increasing certain fibroblast subpopulations, but using a c-Jun inhibitor or fat grafting can reduce this effect.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

M-CSF-stimulated myeloid cells can turn into skin cells and help heal wounds and regrow hair.

The mouse model shows potential for understanding and improving scarless wound healing, and Wnt-4 and TGF-β1 play a role in wound healing and scar formation.

A protein from certain immune cells is key for new hair growth after skin injury in mice.

Mice healed without scars as fetuses but developed scars as adults, suggesting scarless healing might be replicated with further research.

The study found that a specific signaling pathway helps skin wounds heal faster but may lead to larger scars.

The scaffolds significantly sped up wound healing in dogs and were safe.

The study concluded that the new wound model can be used to evaluate skin regeneration and nerve growth.

The document explains how to do skin procedures, care after surgery, and when to use certain treatments.

Dermal stem cells help regenerate hair follicles and heal skin wounds.

MRL/MpJ mice's skin wounds heal with scars, unlike their ear wounds which can regenerate.

Lanyu pigs show that partial-thickness wounds can partially regenerate important skin structures, which may help improve human skin healing.

Damage to skin releases dsRNA, which activates TLR3 and helps in skin and hair follicle regeneration.

Mice without collagen VI have slower hair growth normally but faster regrowth after injury.

Injecting alpha-melanocyte-stimulating hormone in mice improved skin healing and reduced scarring.

Wounds heal without scarring in early development but later result in scars, and studying Wnt signaling could help control scarring.

Wound healing can help prevent hair loss from chemotherapy in young rats by increasing interleukin-1β signaling.

Zinc/silicon-infused hydrogel helps regenerate hair follicles.

Careful selection of mice by genetics and age, and controlled housing conditions improve the reliability of hair regrowth in wound healing tests.

Thymic stromal lymphopoietin (TSLP) promotes hair growth, especially after skin injury.

γδ T cells help with hair growth during wound healing in mice.

Hair growth phase and certain genes can speed up wound healing, while an inflammatory mediator can slow down new hair growth after a wound. Understanding these factors can improve tissue regeneration during wound healing.

Blocking β-catenin in skin cells improves hair growth during wound healing.

Fat stem cells from diabetic mice can still help heal wounds.

New hair can grow from large wounds in mice, but less so as they age, involving reprogramming of skin cells and specific molecular pathways.

P-selectin is not the only factor that prevents scarring in fetal wound healing in mice.

Blood-derived CD34+ cells speed up healing, reduce scarring, and regrow hair in skin wounds.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

Removing REDD1 in mice increases skin fat by making fat cells larger and more numerous.