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Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

Phospholipase A2 enzymes play key roles in skin health and disease.

Boosting β-catenin signaling in certain skin cells can enhance hair growth.

Basonuclin 2 is vital for the development of facial bones, hair follicles, and male germ cells in adult mice, and its absence can lead to dwarfism and abnormal follicles.

EGFR helps control how hair grows and forms without needing p53 protein.

MicroRNAs are important for hair growth regulation, with Dicer being crucial and Tarbp2 less significant.

Autophagy helps keep skin healthy and may improve treatments for skin diseases.

Skin cells show flexibility in healing wounds and forming tumors, with potential for treating hair disorders and chronic ulcers.

Dogs have varying numbers of touch-sensitive Merkel cells in different skin areas, with most in the oral mucosa and facial skin, unrelated to age, sex, breed, or color.

Exosomes could be a promising way to help repair skin and treat skin disorders.

Eicosanoids are crucial for skin health, and targeting their pathways may help treat skin conditions.

Autologous platelet concentrates show promise in esthetic treatments but need more standardized research.

Hair follicle stem cells can form both hair follicles and skin.

Fibroblasts are crucial for tissue repair and inflammation, and understanding them can help treat fibrotic diseases.

We now understand more about what causes acne and this could lead to better, more personalized treatments.

Different small areas within hair follicles send specific signals that control what type of cells stem cells become.

Human skin acts like a hormone-producing organ, making and managing various hormones important for skin and hair health.

EGFR is essential for normal hair development and follicle differentiation.

PPARγ is crucial for skin health but can have both beneficial and harmful effects.

Certain mouse strains develop a skin condition similar to a human hair loss disease due to genetic defects.

Progerin affects cell shape but not hair or skin in mice.

Stem cells, especially from fat tissue and Wharton's jelly, can potentially regenerate hair follicles and treat hair loss, but more research is needed to perfect the treatment.

Odontogenic keratocysts are caused by abnormal Hedgehog signaling and can lead to tooth and bone issues.

The conclusion is that understanding how hair follicle stem cells live or die is important for maintaining healthy tissue and repairing injuries, and could help treat hair loss, but there are still challenges to overcome.

The Msi2 protein helps keep hair follicle stem cells inactive, controlling hair growth and regeneration.

P-cadherin is crucial for hair follicle pigmentation but not skin pigmentation.

Thyroid hormone receptors are essential for hair growth and wound healing.

New treatments for ichthyosis, like protein replacement and gene therapy, show promise and may become standard care.

Increased ODC activity leads to skin tumors by recruiting stem cells, not by toxic byproducts.

The study found that analyzing certain cell signaling pathways is not a reliable method to tell apart two types of skin tumors.