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Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

Thyroid hormone receptors are essential for hair growth and wound healing.

Ornithine decarboxylase is crucial for hair growth and follicle development.

Boosting β-catenin signaling in certain skin cells can enhance hair growth.

PTEN helps control the number and health of skin stem cells by working with the protein BMAL1.

Sox21 is crucial for tooth development and enamel formation by preventing cells from changing into a different type.

Genetic changes in mice help understand skin and hair disorders, aiding treatment development for acne and hair loss.

Alkaline Ceramidase 1 prevents early hair loss in mice by keeping hair follicle stem cells balanced.

MED1 is essential for normal hair growth and maintaining hair follicle stem cells.

Certain daily habits like stress, diet, and sleep can affect the severity of hair loss in alopecia areata.

The KRTAP11-1 gene promoter is crucial for specific expression in sheep wool cortex.

EGFR helps control how hair grows and forms without needing p53 protein.

Improved understanding of stem cell mechanisms can enhance skin tissue engineering.

Knocking out certain genes in mice helps understand skin and hair growth problems.

Non-coding RNAs are important for hair growth and could lead to new hair loss treatments, but more research is needed.

The Bcl-2 protein is important for keeping hair follicle stem cells working and preventing hair loss.

MicroRNAs are important for hair growth regulation, with Dicer being crucial and Tarbp2 less significant.

When skin blisters, healing the wound is more important than growing hair, and certain stem cells mainly fix the blisters without helping hair growth.

Different genes are active in dogs' hair growth and skin, similar to humans, which helps understand dog skin and hair diseases and can relate to human conditions.

RSPO1 mutations in certain patients lead to skin cells that don't develop properly and are more likely to become invasive, increasing the risk of skin cancer.

Thymic stromal lymphopoietin (TSLP) promotes hair growth, especially after skin injury.

Wnt/β-catenin signaling is important for keeping skin cell attachment structures stable.

Fat tissue under the skin affects hair growth and aging; reducing its inflammation may help treat hair loss.

Cell polarity signaling controls tissue mechanics and cell fate, with complex interactions and varying pathways across species.

A faulty KLHL24 gene leads to hair loss by damaging hair follicle stem cells.

Tissue engineering could be a future solution for hair loss, but it's currently expensive, complex, and hard to apply in real-world treatments.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

Keratin protein production in cells is controlled by a complex system that changes with cell type, health, and conditions like injury or cancer.

Touching hair can activate nearby nerve cells through signals from the hair's outer layer.

A specific pathway involving AR, miR-221, and IGF-1 plays a key role in causing common hair loss.