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Male-pattern hair loss is largely influenced by genetics, with key genes identified.

The conclusion is that Treg-targeted therapies have potential, but more knowledge of Treg biology is needed for effective treatments, including for cancer.

The document concludes that understanding how the skin's immune system and inflammation work is complex and requires more research to improve treatments for skin diseases.

Men with less sun-sensitive skin have lower PSA levels, while men with more sun-sensitive skin have higher PSA levels.

T-regulatory cells are important for skin health and can affect hair growth and reduce skin inflammation.

Protein tyrosine kinases are key in male pattern baldness, affecting skin structure, hair growth, and immune responses.

Alopecia areata and vitiligo share immune system dysfunction but differ in specific immune responses and affected areas.

Using radiation to make mice's hair turn gray helps study and find ways to prevent or reverse hair graying.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

The document concludes that understanding hair follicles requires more research using computational methods and an integrative approach, considering the current limitations in hair treatment products.

Premature hair greying may indicate a higher risk of metabolic problems.

Some viruses can cause cancer by changing cell processes and avoiding the immune system; vaccines and targeted treatments help reduce these cancers.

Researchers found 15 new genetic links to skin traits in Japanese women.

Skin stem cells are crucial for maintaining and repairing the skin and hair, using a complex mix of signals to do so.

Plant extracts may help prevent or reverse hair graying.

The protein STAT3 slows down cell growth by blocking the FST gene, which affects hair development in sheep.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

Alopecia areata, a type of hair loss, is likely an autoimmune disease with a genetic link, but its exact cause is still unknown.

High levels of ERK activity are key for tissue regeneration in spiny mice, and activating ERK can potentially redirect scar-forming healing towards regenerative healing in mammals.

The JAK/STAT pathway is important in cell processes and disease, and JAK inhibitors are promising for treating related conditions.

Black cumin and its nanoformulations show promise in treating neurodegenerative diseases.

The research suggests immune system changes and specific gene expression may contribute to male hair loss, proposing potential new treatments.

TNFα, IFNγ, and Substance P significantly affect prolactin levels in human skin, suggesting new treatments for skin and hair conditions.

Key genes linked to immune response are upregulated in hair follicles and skin tissues in chronic discoid lupus erythematosus.

Alopecia areata is caused by immune system issues, and JAK inhibitors might help treat it.

CO2 laser treatment for aging skin significantly changes immune system-related genes.

Key genes linked to immune response are highly active in lupus-affected hair follicles.

A substance called poly(I:C) increases a protein called carbonic anhydrase II in skin cells, which might help with skin defense and healing.

The TGF-β family helps control how cells change and move, affecting skin, hair, and organ development.

Ovol2 is crucial for hair growth and skin healing by controlling cell movement and growth.