Search

everythinglearnresearchcommunity

for

Search:↓

New insights into cell communication in psoriasis suggest innovative drug treatments.

Different immune responses cause hair loss in scalp diseases, with unique patterns in scalp psoriasis possibly protecting against hair loss.

Stress may contribute to hair loss in alopecia areata by affecting immune responses and cell death in hair follicles.

The protein EZH2 blocks microRNA-22, increasing STK40 protein, which helps hair follicle stem cells change and grow hair.

Cell death shapes skin stem cell environments, affecting inflammation, repair, and cancer.

IL-33 is linked to hair follicle damage in psoriasis and could be a treatment target for hair loss in this condition.

CRH in the skin acts like the body's stress response system, affecting cell behavior and immune activity.

Parathyroid hormones are important for hair growth, but their use in treating hair loss from chemotherapy is still uncertain.

Blocking Wnt/β-catenin signaling with EGF receptor is necessary for proper hair growth.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Th22 cells are essential for Tβ15-induced hair growth in mice.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

T-regulatory cells are important for skin health and can affect hair growth and reduce skin inflammation.

Skin bacteria, specifically Staphylococcus aureus, help in wound healing and hair growth by using IL-1β signaling. Using antibiotics on skin wounds can slow down this natural healing process.

Plant adaptogens may help treat skin diseases and improve skin health, but more research is needed to fully understand how they work.

Glutamic acid helps increase hair growth in mice.

Blocking JAK-STAT signaling can lead to hair growth.

Alopecia areata is an autoimmune disease causing patchy hair loss, often with other autoimmune disorders, but its exact causes are unknown.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

New treatments for vitiligo may focus on protecting melanocyte stem cells from stress and targeting specific pathways involved in the condition.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Certain types of T cells are essential for healthy skin and play a role in skin diseases, but more research is needed to improve treatments.

The JAK/STAT pathway is important in cell processes and disease, and JAK inhibitors are promising for treating related conditions.

Tcf3 helps cells move and heal wounds by controlling lipocalin 2.

The trial will test if YH0618 granule prevents hair loss in breast cancer patients during chemotherapy.

Skin cell-derived vesicles can help heal skin injuries effectively.

No cure exists for alopecia areata, and treatments are personalized.

The health of the gut may be important in developing new ways to prevent, diagnose, and treat alopecia areata.

Certain natural and synthetic compounds may help treat inflammatory skin diseases by targeting a specific signaling pathway.

Hair follicles are hormone-sensitive and involved in growth and other functions, with potential for new treatments, but more research is needed.