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Two specific hair keratin genes are active during hair growth and decline as hair transitions to rest.

SOX2 is crucial for skin cell function and hair growth, and it plays a role in skin cancer and wound healing.

Skin cysts might help advance stem cell treatments to repair skin.

Removing centrosomes from skin cells leads to thinner skin and stops hair growth, but does not greatly affect skin cell differentiation.

Rac1 is essential for proper hair structure and color.

Cytokeratin 19 and cytokeratin 15 are key markers for monitoring the quality and self-renewing potential of engineered skin.

Certain skin cells near the base of hair muscles may help renew and stabilize skin, possibly affecting skin disorder understanding.

A specific group of slow-growing stem cells marked by Thy1 is crucial for skin maintenance and healing in mice.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

Human skin cells with stem-like features can help create new hair follicles and sebaceous glands when combined with other cells.

K42 and K124 keratins are only found in horse hoof lamellae.

Skin cells show flexibility in healing wounds and forming tumors, with potential for treating hair disorders and chronic ulcers.

Epidermal keratinocytes start wound healing and inflammation after schistosome infection.

MOF controls skin development by regulating genes for mitochondria and cilia.

MOF controls key genes for skin development by regulating mitochondrial and ciliary functions.

The research identified two types of keratinocytes in chicken scales: one for hard scales and another for soft skin, with similarities to human skin differentiation.

CD4+ skin cells may be precursors to basal cell carcinoma.

The method increases stem-like cells for better skin regeneration.

The conclusion is that the nuclear lamina and LINC complex in skin cells respond to mechanical signals, affecting gene expression and cell differentiation, which is important for skin health and can impact skin diseases.

SOX9 helps determine stem cell roles by interacting with DNA and proteins that control gene activity.

Skin spheroids with both outer and inner layers are key for regrowing skin patterns and hair.

The document concludes that skin stem cells are important for hair growth and wound healing, and could be used in regenerative medicine.

Dandruff might be caused by changes in how hair follicles naturally release oils and an immune response to this imbalance.

Mirex seems to promote a unique group of skin cells different from those affected by another tumor promoter, TPA.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

MicroRNAs are crucial for controlling skin development and healing by regulating genes.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

Growth factors are crucial for hair development and could help treat hair diseases.

PDCD4 is important for controlling skin cell growth and healing.