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The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Melatonin helps goat hair stem cells grow and maintain their ability to become different cell types.

A specific RNA increases hair stem cell growth and skin healing by affecting a protein through interaction with a microRNA.

LGR5 is a common marker of hair follicle stem cells in different animals and is important for hair growth and regeneration.

Different genes are active in dogs' hair growth and skin, similar to humans, which helps understand dog skin and hair diseases and can relate to human conditions.

New research shows that skin diversity is influenced by different types of dermal fibroblasts and their development, especially involving the Wnt/β-catenin pathway.

Eyelid cells share signaling components but differ in pathway activity.

Overactive Wnt signaling in mouse skin stem cells causes acne-like cysts and shrinking oil glands, which some treatments can partially fix.

Aging causes hair loss and graying due to stem cell decline and changes in cell behavior and communication.

The document concludes that various signaling pathways and genetic factors are crucial for chicken feather development, affecting poultry quality.

The enzymes Tet2 and Tet3 are important for skin cell development and hair growth.

Ptch2 plays a key role in controlling stem cell function and the ability to regenerate after birth.

The protein STAT3 slows down cell growth by blocking the FST gene, which affects hair development in sheep.

Certain gene variations in the Vitamin D Receptor are linked to higher risk of female hair loss.

Disrupted stem cell signals in hairpoor mice cause hair loss.

miR-199a-3p controls hair growth and is linked to alopecia areata.

Keeping β-catenin levels high in mammary cells disrupts their development and branching.

A protein called desmoglein 3 is important for keeping hair follicle stem cells inactive and helps in their regeneration.

Removing Dicer from pigment cells in newborn mice causes early hair graying and changes in cell migration molecules.

Hair loss from Alopecia Areata is caused by both genes and environment, with several treatments available but challenges in cost and relapse remain.

CRABP2 helps increase the growth of cells important for hair growth by activating a specific growth pathway.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

Wnt4 protein makes the outer skin layer thicker in burn wounds by turning on a specific healing pathway and loosening the connections between skin cells.

Understanding how androgens and their receptors work can lead to improved treatments for skin diseases.

Vitamin D receptor is crucial for skin wound healing.

Enterococcus faecalis delays wound healing by disrupting cell functions and creating an anti-inflammatory environment.

Gynostemma pentaphyllum and its component damulin B could help hair grow by activating certain cell pathways.

Hair loss in Androgenetic Alopecia is caused by genetics, aging, and lifestyle, leading to hair follicle shrinkage and related health risks.

Substances from fat-derived stem cells can promote hair growth and counteract hormone-related hair loss by activating a key hair growth pathway.

Turning off the Lhx2 gene in mouse embryos leads to slower wound healing and scars.