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Retinoic acid production and signaling in hair follicles are regulated by location and timing, affecting hair growth and cycling.

Noncoding dsRNA boosts hair growth by activating TLR3 and increasing retinoic acid.

Retinoic acid, glucocorticoids, and IGF1 increase IGFBP-3 production in human dermal papilla cells, affecting hair growth.

The enzymes Aldh1a2 and Aldh1a3 are involved in making retinoic acid in hair follicles and have different roles in hair growth.

Retinoic acid is effective for treating eruptive vellus hair cysts.

Activating TLR3 may help produce retinoic acid, important for tissue regeneration.

Oral retinoic acid effectively treated collodion baby, with hair loss as the main side effect.

Hair follicle stem cells can become nerve cells using specific treatments.

Retinoic acid is not linked to hair loss from fluconazole use.

High concentrations of retinoic acid kill hair follicle stem cells, while low concentrations may help treat skin diseases.

The CRABP I gene in cashmere goats is highly conserved but has unique features at specific amino sites.

Non-coding RNA boosts retinoic acid production and signaling, aiding regeneration.

Double-stranded RNA helps regenerate hair follicles by increasing retinoic acid production and signaling.

Retinoic acid affects hair growth and can cause hair loss.

Minoxidil and ATRA together boost hair growth more effectively than minoxidil alone.

Some vitamin D analogs can thicken skin and reduce pore size like a common acne treatment, with one analog also affecting skin growth factors.

SMR-2 and SMR-6 are much more toxic than retinoic acid, causing severe side effects.

BMPR1a-ECD reduces wrinkles much more effectively than retinoic acid.

Minoxidil is the main over-the-counter treatment for hair loss, with other options like finasteride, latanoprost, retinoic acid, antioxidants, biotin, nutraceuticals, and platelet-rich plasma therapy also showing promise.

A new method allows for controlled, long-lasting delivery of retinoic acid through the skin with less inflammation.

SMR-2 and SMR-6 are much more toxic than retinoic acid, causing severe symptoms and organ damage in mice.

ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

The document concludes that recent research has improved understanding of skin diseases and the balance between cell growth and differentiation in the epidermis.

Scientists turned mouse stem cells into skin cells that can grow into skin layers and structures.

Different skin treatments work best for different levels of wrinkles and sun damage.

Hormones control keratin production in skin and hair.

Researchers found a way to create cells from stem cells that act like human cells important for hair growth and could be used for hair regeneration treatments.

Hair can regrow in large wounds through a process similar to how hair forms in embryos, and understanding this could lead to new treatments for hair loss or scarring.

Shaving can cause a viral skin infection that looks like bacterial folliculitis.

Fluorescence can effectively measure acne treatment progress.