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The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

Dermal EZH2 controls skin cell growth and differentiation in mice.

A gene mutation causes early keratinocyte maturation leading to hair loss in Olmsted syndrome.

Keratinocytes and adipose-derived stem cells can effectively heal difficult skin wounds.

PDCD4 is important for controlling skin cell growth and healing.

Ezh2 controls skin development by balancing signals for dermal and epidermal growth.

The skin's layers protect, sense, and regulate the body's internal balance, but can be prone to cancer.

Activating Nrf2 helps wounds heal faster by increasing hair follicle stem cells.

WWOX deficiency in mice causes skin and fat tissue problems due to disrupted cell survival signals.

Scaffoldin helps form hard skin structures in chicken embryos.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Autophagy changes the protein makeup of hair.

Radiation therapy in breast cancer patients changes gene expression related to DNA damage, fibroblast growth, and hair follicle development, which could help improve treatment for radiation-induced fibrosis.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.

Changes in the SREBF1 gene cause a rare genetic skin and hair disorder.

Understanding keratinization is crucial for treating skin conditions like ichthyoses and psoriasis.

Bird foot scales develop differently and can repair but not fully regenerate due to the lack of specialized stem cell areas.

Transglutaminase activity is important for skin and is found in both mammals and birds.

A single medium, PRIME AIRLIFT, supports better human hair follicle formation in grafts.

Wnt/β-catenin signaling is important for keeping skin cell attachment structures stable.

A specific gene mutation causes Olmsted syndrome.

Cystatin M/E strongly inhibits cathepsin V and cathepsin L, important for skin formation.

Cell death shapes skin stem cell environments, affecting inflammation, repair, and cancer.

Anti-acne medications may work by reducing the activity of a protein involved in acne development.

In situ DNA labeling in hair can help predict forensic DNA analysis success.

Over 67 genes linked to ichthyosis help improve diagnosis and treatment.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

A new mutation in the ST14 gene causes a rare skin and hair disorder in a specific family.

The cornified envelope is crucial for skin's barrier function and involves key proteins and genetic factors.

Drug repositioning offers hope for new, affordable treatments for a genetic skin disorder called ARCI.