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The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

Exosomes show promise for tissue repair and regeneration with advantages over traditional cell therapies.

The document lists various dermatology topics, treatments, and diagnostic methods.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

Activin A helps heal skin wounds and protects the brain after injury.

Canine hair follicles have stem cells similar to human hair follicles, useful for studying hair disorders.

Colorectal cancer development involves both genetic changes and epigenetic alterations like DNA methylation and microRNA changes.

Blocking testosterone speeds up wound healing in males.

cDermo-1 causes dense skin, feathers, and scales in chickens.

Blocking testosterone speeds up wound healing in males.

A protein called sFRP4 can slow down hair regrowth.

High wool density in Rex rabbits is linked to specific gene activity affecting hair follicle development.

The protein STAT3 slows down cell growth by blocking the FST gene, which affects hair development in sheep.

High levels of ST14 and TMEFF1 proteins in ovarian cancer are linked to worse patient outcomes and may be a new treatment target.

Understanding hair follicle development can help treat hair loss, skin regeneration, and certain skin cancers.

TGF-β2 helps activate hair follicle stem cells by counteracting BMP signals.

Men with baldness due to androgenetic alopecia still have hair stem cells, but lack specific cells needed for hair growth.

Hair growth and development are controlled by specific signaling pathways.

Ectodysplasin inhibits Wnt signaling to help form hair follicles.

Feather patterns form through genetic and epigenetic controls, with cells self-organizing into periodic patterns.

High LGR5 levels in glioblastoma indicate poor prognosis and are essential for cancer stem cell survival.

Sostdc1 controls the size and number of hair and mammary gland structures.

Noggin gene inactivation causes skeletal defects in mice, varying by genetic background.

Odontogenic keratocysts are caused by abnormal Hedgehog signaling and can lead to tooth and bone issues.

The document concludes that more multidisciplinary research is needed to understand and treat PCOS, a condition that significantly affects women's health and quality of life.

Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

Sox21 is crucial for tooth development and enamel formation by preventing cells from changing into a different type.

A certain signaling pathway in mice, when increased, causes hair to gray by depleting the cells that give hair its color.

Bmpr2 and Acvr2a receptors are crucial for hair retention and color.

Knocking out certain genes in mice helps understand skin and hair growth problems.