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Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

The TGF-β family helps control how cells change and move, affecting skin, hair, and organ development.

Wnt signaling may be linked to heart diseases in aging and could be a target for future treatments.

The Wnt/β-catenin pathway is important for tissue development and has potential in regenerative medicine, but requires more research for therapeutic use.

Wnt3a activates certain genes in hair follicle cells, including a newly discovered one, EP2, which may affect hair growth.

The PDGF/PDGFR pathway is a potential drug target with mixed success in treating various diseases, including some cancers and fibrosis.

Blocking Wnt signaling in young mice causes thymus shrinkage and cell loss, but recovery is possible when the block is removed.

Editing the FGF5 gene in sheep increases fine wool growth.

FGF9 helps hair follicles grow in small-tailed Han sheep by affecting cell growth and certain signaling pathways.

Increased FGFR2b signaling, influenced by androgens, plays a role in causing acne.

BMP signaling controls hair follicle size and cell growth by affecting cell cycle genes.

Male genital development is driven by androgen signaling and understanding it could help address congenital anomalies.

Gab1 protein is crucial for hair growth and stem cell renewal, and Mapk signaling helps maintain these processes.

Stem cell niches are crucial for regulating stem cell renewal and differentiation, and understanding them can help in developing regenerative therapies.

Hair stem cell regeneration is controlled by signals that can explain different hair growth patterns and baldness.

3,4,5-tri-O-caffeoylquinic acid promotes hair growth by activating the β-catenin pathway.

Olive leaf compound oleuropein helps grow hair in mice.

Proper control of β-catenin activity is crucial for development and preventing diseases like cancer.

Modified stem cells from umbilical cord blood can make hair grow faster.

The conclusion is that teeth, hair, and claws have similar stem cell niches, which are important for growth and repair, and more research is needed on their regulation and potential markers.

Increasing Wnt10b levels can help grow new hair follicles in mice.

New research shows that skin diversity is influenced by different types of dermal fibroblasts and their development, especially involving the Wnt/β-catenin pathway.

Mice can grow new hair follicles after skin wounds through a process not involving existing hair stem cells, but requiring more research to understand fully.

Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

The PP2A-B55α protein is essential for brain and skin development in embryos.

Increasing alkaline phosphatase in human skin cells helps to grow more hair.

The research identified genes that explain why some sheep have curly wool and others have straight wool.

Skin development in mammals is controlled by key proteins and signals from underlying cells, involving stem cells for maintenance and repair.

Skin spheroids with both outer and inner layers are key for regrowing skin patterns and hair.

lncRNAs may regulate hair follicle development in Hu sheep.