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Rats can't grow new hair follicles after skin wounds, unlike mice, due to differences in gene expression and response to WNT signaling.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

Thyrotropin-Releasing Hormone helps heal wounds in frog and human skin.

Skin cells show flexibility in healing wounds and forming tumors, with potential for treating hair disorders and chronic ulcers.

Activin activation in skin cells speeds up wound healing without affecting scar quality.

Different types of stem cells in hair follicles play unique roles in wound healing and hair growth, with some stem cells not originating from existing hair follicles but from non-hair follicle cells. WNT signaling and the Lhx2 factor are key in creating new hair follicles.

Activin helps skin growth and healing mainly through stromal cells and affects keratinocytes based on its amount.

The gene Msx2 is crucial for hair follicle regeneration during wound healing.

Adult mice can grow new hair from skin wounds.

Skin and hair renewal is maintained by both fast and slow cycling stem cells, with hair regrowth primarily driven by specific stem cells in the hair follicle bulge. These cells can also help heal wounds and potentially treat hair loss.

Both cell and non-cell parts are important for rat whisker follicle regrowth.

Hair can regrow in large wounds through a process similar to how hair forms in embryos, and understanding this could lead to new treatments for hair loss or scarring.

Lizards can regrow their tail scales with the same structure, distribution, and gender-specific features as the original ones, and this unique ability is not seen in adult mammals.

Hair growth phase and certain genes can speed up wound healing, while an inflammatory mediator can slow down new hair growth after a wound. Understanding these factors can improve tissue regeneration during wound healing.

Tissue stiffness affects hair follicle regeneration, and Twist1 is a key regulator.

MED1 affects skin wound healing differently in young and old mice.

Fractional photothermolysis helps wounds heal with minimal scarring.

Stromal stem cells may help heal wounds by becoming structural cells or affecting the immune system, but more research is needed to understand how.

The Sonic hedgehog pathway is crucial for new hair growth during mouse skin healing.

Fetal wound healing changes with development, affecting inflammation and collagen, which may influence scarring.

Mice can grow new hair follicles after skin wounds through a process not involving existing hair stem cells, but requiring more research to understand fully.

Regulatory T cells are essential for normal and improved wound healing in mice.

BRG1 is essential for skin cells to move and heal wounds properly.

The African spiny mouse can fully regenerate its muscle without scarring, unlike the common house mouse.

Dermal stem cells help regenerate hair follicles and heal skin wounds.

A new hydrogel with micronized amnion helps achieve better, scar-free skin healing.

The research found that different types of fibroblasts are involved in wound healing and that some blood cells can turn into fat cells during this process.

Activating Nrf2 in skin cells speeds up wound healing by increasing the growth of certain stem cells.

The combined treatment with engineered bacteria and yellow LED light improved wound healing in mice.

Careful selection of mice by genetics and age, and controlled housing conditions improve the reliability of hair regrowth in wound healing tests.