Keratinocyte-Derived Follistatin Regulates Epidermal Homeostasis and Wound Repair

    December 2008 in “ Laboratory investigation ”
    Maria Antsiferova, Jennifer E. Klatte, Enikõ Bodó … Heidi Kögel
    Studysummary This study observed that selectively activating activin in the epidermis of mice enhanced reepithelialization during wound healing without altering the quality of the healed wound.
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    Research cited in this study 5

    1. Activin Controls Skin Morphogenesis and Wound Repair Predominantly via Stromal Cells and in a Concentration-Dependent Manner via Keratinocytes ˜The œAmerican journal of pathology · 2005
    2. Control of Pelage Hair Follicle Development and Cycling by Complex Interactions Between Follistatin and Activin The FASEB Journal · 2003
    3. The Activin Binding Proteins Follistatin and Follistatin-Related Protein Are Differentially Regulated In Vitro and During Cutaneous Wound Repair Journal of Endocrinology/Journal of endocrinology · 2001
    4. Impaired Wound Healing in Transgenic Mice Overexpressing the Activin Antagonist Follistatin in the Epidermis EMBO journal · 2001
    5. Overexpression of Activin A in the Skin of Transgenic Mice Reveals New Activities in Epidermal Morphogenesis, Dermal Fibrosis, and Wound Repair EMBO journal · 1999

    Related research 1

    1. Hair Growth Control by Follistatin Enshou saisei · 2004