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Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.

The mouse hairy ears mutation causes longer ear hair due to changes in gene expression.

Radiation significantly slows down wound healing in mice.

Frizzled 3 and Frizzled 6 together control the orientation of mouse hair follicles.

Scientists have made progress in understanding hair follicle stem cells, identifying specific genes and markers, and suggesting their use in treating hair and skin conditions.

Hair growth environment recreated with challenges; stem cells make successful skin organoids.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

Glucocorticoids can cause various health issues, but some treatments may help reduce these effects.

Researchers found new genes involved in hair growth, which could help develop new hair treatments.

UV exposure reduces Lgr6+ stem cells in mouse skin and they don't significantly contribute to skin cancer development.

Gene therapy in mice increased lifespan and improved health without causing cancer.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

sPLA2-IIE is crucial for normal hair follicle structure and skin health.

A 3-month stay in space causes skin thinning, disrupts hair growth, and changes muscle-related genes in mice.

Some treatments can improve skin's defense against damage, but overuse may cause other skin problems.

Deleting the p63 gene in certain cells causes problems in thymus development and severe hair loss in mice.

New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

Tiny pollution particles called PM2.5 can harm skin cells by causing stress, damage to cell parts, and cell death.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

The document concludes that understanding hair and feather regeneration can help develop new regenerative medicine strategies.

Fibromodulin treatment helps reduce scarring and improves wound healing by making it more like fetal healing.

Different sPLA2 enzymes affect immunity, skin and hair health, reproduction, and may be potential targets for therapy.

Applying a special DNA plasmid to the skin can make it thicker and stronger.

Using skin stem cells and certain molecules might lead to scar-free skin healing.

Putting thymidine dinucleotide on newborn mice's skin can delay and reduce skin cancer.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Stress increases a factor in mice that leads to hair loss, and blocking this factor may prevent it.

Activating the Wnt/β-catenin pathway could lead to new hair loss treatments.

TCDD speeds up skin barrier formation by increasing certain gene expressions.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.