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A hormone affects hair growth, certain molecules may prevent skin damage, a skin disease is linked to immune cells, glycerol helps skin hydration, and psoriasis treatment trials need improvement.

ANp63 is crucial for skin integrity, new filaggrin gene mutations link to eczema, hair can regrow from non-stem cells, sunburns are increasing, and glucocorticoids help treat skin allergies by affecting immune cells.

The immune system plays a key role in tissue repair, affecting both healing quality and regenerative ability.

Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

Skin grafts on mice can cause an immune response leading to hair loss, useful for studying human hair loss conditions.

Substances from Chinese medicines show promise for immune support and disease prevention, but the way they are processed affects their effectiveness.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

Alopecia areata is influenced by genetics and immune system factors, and better understanding could improve treatments.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

Healthy mitochondria in skin cells are essential for proper hair growth and skin cell interaction in mice.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.

RXR-alpha is strongly expressed in both normal and psoriatic skin and may help in skin cell differentiation and hair growth.

Genetic variants linked to ten skin diseases were found, showing both immune and non-immune factors play a role.

Skin organoids from stem cells could better mimic real skin but face challenges.

Different parts of the body have unique immune characteristics in their skin.

Alopecia areata involves persistent gene abnormalities and immune activity, even in regrown hair, suggesting a risk of relapse.

The document concludes that immune system abnormalities cause alopecia areata, but the exact process is still not completely understood.

The research confirms that Hidradenitis Suppurativa is characterized by increased inflammation, disrupted skin cell organization, and abnormal metabolic processes.

Skinny fat cells help wounds heal faster by releasing fatty acids.

Alopecia areata is caused by immune system issues, and JAK inhibitors might help treat it.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

The document concludes that the immune-inhibitory environment of the hair follicle may prevent melanoma development.

Autoimmune reactions may cause both alopecia areata and HAM.

Talc in street cocaine can cause immune-reactive skin nodules where injected.

Autoimmune hair loss may be linked to increased Th1 and Th17 cells and decreased Th2 cells.

Poor response to topical immunotherapy in alopecia areata patients is linked to impaired cell responses.

Implanting hair-follicle stem cells in mice brains helped repair brain bleeding and reduced brain inflammation.

The cause of Frontal fibrosing alopecia, a type of hair loss, is complex, likely involving immune responses and genetics, but is not fully understood.