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Scientists have made progress in understanding hair follicle stem cells, identifying specific genes and markers, and suggesting their use in treating hair and skin conditions.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.

Mouse profilaggrin helps in skin cell differentiation and may be involved in calcium signaling.

Understanding where cancer cells come from helps create better prevention and treatment methods.

EZH2 is essential for hair growth and skin cell development.

Mitochondrial reactive oxygen species are essential for skin and hair development.

CD4+ skin cells may be precursors to basal cell carcinoma.

EAAT4 decreases with age, harming skin function and calcium balance.

Cornification is the process where living skin cells die to create a protective barrier, and problems with it can cause skin diseases.

Overexpressing Dsg3 in mice skin causes excessive cell growth and abnormal skin development.

EGFR is essential for normal hair development and follicle differentiation.

The TGM3 gene's promoter region is key for skin and hair cell function and may aid gene therapy.

PPARγ is essential for maintaining healthy skin, controlling inflammation, and ensuring proper skin barrier function.

The research suggests new treatments for skin cancer could target specific cell growth pathways.

Vitamin D helps skin stem cells heal wounds by working with a key skin protein.

Epigenetic mechanisms control skin development by regulating gene expression.

Adult stem cells from rat whisker follicles can regenerate hair follicles and sebaceous glands.

Epigenetic changes control how adult stem cells work and can lead to diseases like cancer if they go wrong.

Wounded skin cells can revert to stem cells and help heal.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

The TRPV3 gene mutation affects hair growth by keeping mice in the growth phase longer, which could help treat hair loss.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

Heparan sulfate is important for hair growth, preventing new hair formation in mature skin, and controlling oil gland development.

GRHL3 is important for controlling gene activity in skin cells during different stages of their development.

Blocking Wnt/β-catenin signaling with EGF receptor is necessary for proper hair growth.

The study found key factors in the cause of hidradenitis suppurativa, its link to other diseases, and identified existing drugs that could potentially treat it.

Key genes crucial for sheep hair follicle development were identified, aiding fine wool breeding and human hair loss research.

The study created hair-bearing skin models that lack a key protein for skin layer attachment, limiting their use for certain skin disease research.

Vitamin D and calcium are important for skin stem cell function and wound healing.

Glutamic acid helps increase hair growth in mice.