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The enzyme DHHC13 is essential for healthy hair and skin, and its deficiency leads to hair loss and skin problems.

A gene mutation in mice causes skin defects and early death.

Cornification is the process where living skin cells die to create a protective barrier, and problems with it can cause skin diseases.

Cystatin M/E strongly inhibits cathepsin V and cathepsin L, important for skin formation.

Different types of cell death affect skin health and inflammation, and understanding them could improve treatments for skin diseases.

Cornification is how skin cells die to form the protective outer layer of skin, hair, and nails.

Epigenetic mechanisms control skin development by regulating gene expression.

The research identified two types of keratinocytes in chicken scales: one for hard scales and another for soft skin, with similarities to human skin differentiation.

Lack of cystatin M/E causes thin hair and dry skin.

Understanding proteins in skin structures like claws and hair is crucial for future research.

PPARγ is essential for maintaining healthy skin, controlling inflammation, and ensuring proper skin barrier function.

TCDD speeds up skin barrier formation by increasing certain gene expressions.

The S100A4 protein is more common in psoriatic skin and could be a target for treating psoriasis.

Nagashima-type palmoplantar keratosis in Asians is caused by a SERPINB7 gene mutation.

TGM3 is important for skin and hair structure and may help diagnose cancer.

Autophagy changes the protein makeup of hair.

Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

Long non-coding RNAs help regulate wool fineness in Gansu alpine fine-wool sheep.

RSPO1 mutations in certain patients lead to skin cells that don't develop properly and are more likely to become invasive, increasing the risk of skin cancer.

The hair follicle infundibulum plays a key role in skin health and disease, and understanding it better could lead to new skin disease treatments.

TGase 3 helps build hair structure by forming strong bonds between proteins.

Changes in the SREBF1 gene cause a rare genetic skin and hair disorder.

Pangolins have lost some skin-related genes, but kept others, leading to their unique scales and skin features.

The research confirms that Hidradenitis Suppurativa is characterized by increased inflammation, disrupted skin cell organization, and abnormal metabolic processes.

ZDHHC13 is important for normal liver function and metabolism, affecting mitochondrial activity.

S100A3 protein is crucial for hair shaft formation in mice.

Targeting the IL-23/IL-17 pathway effectively treats several inflammatory skin diseases.

Faulty inflammasome activation may lead to autoimmune skin diseases and could be a target for new treatments.

Keratin 79 marks a new group of cells that are key for creating and repairing the hair follicle's structure.