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The reconstructed skin model from hair follicles functions like human skin in processing chemicals and can be used to test ingredient safety.

Exosomes from dermal papilla cells help hair growth by making hair follicle stem cells multiply and change.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

Hair loss is mainly caused by hormones, autoimmune issues, and chemotherapy, and needs more research for treatments.

Betamethasone dipropionate reduced skin thickness, fish oil increased it, and combined treatment had no significant effect.

Knocking out certain genes in mice helps understand skin and hair growth problems.

Blocking certain immune signals can reduce skin damage from radiation therapy.

Markers CRABP1, Nestin, and Ephrin B2 are present in skin cancer environments and may influence their development.

BMAL1 controls skin cell growth and UV damage risk, peaking at night.

ERULUS controls root hair growth by regulating cell wall composition and pectin activity.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

Mice with too much Claudin-6 have skin barrier problems and abnormal hair growth.

UVB radiation harms hair growth and health, causing cell death and other changes in human hair follicles.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

The conclusion is that we need more effective hair loss treatments than the current ones, and these could include new drugs, gene and stem cell therapy, hormones, and scalp cooling, but they all need thorough safety testing.

Fat tissue extract may help treat vitiligo by reducing cell stress and promoting skin repair.

Low-dose radiation affects hair stem cell function and survival by changing their genetic material's structure.

Melatonin affects cashmere growth in goats by influencing stem cell and certain signaling pathways.

Stem cells could potentially rebuild missing structures in wounds, improving facial skin replacement techniques.

New nanofiber technology improves wound healing by supporting cell growth and delivering treatments directly to the wound.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

Fibroblast growth factor receptor signaling controls cell development and repair, and its malfunction can cause disorders and cancer, but it also offers potential for targeted therapies.

Certain immune cells in the skin release a protein that stops hair growth by keeping hair stem cells inactive.

Cell-based therapies using dermal papilla cells and adipocyte lineage cells show potential for hair regeneration.

Minoxidil affects cell growth in two ways: low doses increase growth, while high doses slow it down and can be toxic.

BMP signaling is essential for the development of touch domes.

Advanced therapies like gene, cell, and tissue engineering show promise for hair regrowth in alopecia, but their safety and effectiveness need more verification.

Dexpanthenol helps human hair follicle cells grow by preventing aging and death, and by supporting growth signals.

Cell transplantation faces challenges in genitourinary reconstruction, but alternative tissue sources and microencapsulation show promise.

Mice with damaged skin or hair follicles are more susceptible to anthrax infection.