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Removing Mediator 1 causes teeth cells to turn into hair cells.

Super-enhancers controlled by pioneer factors like SOX9 are crucial for stem cell adaptability and identity.

Removing Mediator 1 from certain mouse cells causes teeth to grow hair instead of enamel.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

The document concludes that understanding hair follicle cell cycles is crucial for hair growth and alopecia research, and recommends specific techniques and future research directions.

Different stem cells are key for hair growth and health, and understanding their regulation could help treat hair loss.

Different signals work together to change gene activity and guide hair follicle stem cells to become specific cell types.

Enhancers and super-enhancers are key in controlling specific gene activity and can play a role in cancer development.

Epigenetic factors play a crucial role in skin health and disease.

MicroRNAs are crucial for skin development, regeneration, and disease treatment.

The study showed that hair follicle stem cells can maintain and organize themselves in a lab setting, keeping their ability to renew and form hair and skin.

Hair can regrow after certain stem cells are lost because other stem cells can take over their role.

GRHL3 is important for controlling gene activity in skin cells during different stages of their development.

Different human hair follicle stem cells grow at different rates and respond differently to a baldness-related compound.

Certain signals are important for reducing specific chemical markers on hair follicle stem cells during rest periods, which is necessary for healthy hair growth.

Ovol2 is crucial for hair growth and skin healing by controlling cell movement and growth.

Skin cells show flexibility in healing wounds and forming tumors, with potential for treating hair disorders and chronic ulcers.

Enzymes called PADIs play a key role in hair growth and loss.

PDGFA/AKT signaling is important for the growth and maintenance of certain skin fat cells.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

Ruxolitinib helps protect skin stem cells and keeps skin healthy in mice with skin GVHD.

Hair follicles are valuable for regenerative medicine and wound healing.

Foxn1 gene regulation is crucial for thymus development but not for hair growth.

Different small areas within hair follicles send specific signals that control what type of cells stem cells become.

Vitamin A helps skin stem cells decide their function, aiding in hair growth and wound repair.

Exosomes from dermal papilla cells help hair growth by making hair follicle stem cells multiply and change.

Polycomb Repressive Complex 2 is not needed for hair regeneration.

Epidermal stem cells help heal severe skin wounds and have potential for medical treatments.

The Ovol2-Zeb1 circuit is crucial for skin healing and hair growth by guiding cell movement and growth.