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Certain gene mutations in Japanese people are linked to different types of hair loss, with some causing mild hair thinning and others leading to complete baldness.

A genetic mutation in the LIPH gene causes hair loss and growth defects.

The first Japanese case of a genetic hair disorder caused by specific mutations in the LIPH gene was identified.

Activating c-Myc in skin causes rapid cell growth and changes, but these effects are reversible.

c-Myc activation in mouse skin increases sebaceous gland growth and affects hair follicle development.

Cannabinoids may help treat various skin conditions.

Wnt and Notch signaling help wound healing by promoting cell growth and regulating cell differentiation.

A new mutation in the human glucocorticoid receptor reduces its function and causes resistance to glucocorticoids.

The balance between androgen receptor and p53 is crucial for sebaceous gland differentiation.

C/EBPalpha and C/EBPbeta are crucial for normal skin and oil gland cell development in adult mice.

Different Myc family proteins are located in various parts of the hair follicle and may affect stem cell behavior.

Stress from being transgender is linked to higher blood pressure at night and more inflammation, which may affect heart health.

SCF and c-Kit decrease in AGA hair follicles, possibly affecting hair pigmentation and growth.

ALA-PDT is effective and safe for chronic X-ray dermatitis, providing complete or partial remission.

Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

ISCK03 stops melanin production in human melanoma cells and lightens skin color in mice and guinea pigs.

C-reactive protein helps monocytes stick to blood vessel cells by causing oxidative stress.

Higher hs-CRP levels in Indian adolescent women with PCOS are more related to BMI than PCOS itself.

Different halogens on progesterone derivatives can either block or mimic male hormone effects, depending on their type and amount.

DA-9401 helps protect rat testis from finasteride damage.

Certain proteins and their receptors are more active during the growth phase of human hair and could be targeted to treat hair disorders.

Hair loss in certain young mice is linked to a specific gene and can be caused by lack of iron.

A specific gene mutation in Japanese people can cause varying degrees of hair thinning in adulthood.

The C-terminal tail of AHF/trichohyalin is essential for organizing keratin filaments in keratinocytes.

DHT affects bone growth by altering gene activity in osteoblasts, potentially complicating steroid use.

c-Myc, Max, and Bin1 help hair follicle cells mature and die.

Glypican-1 is important for blood vessel growth in hair follicles and could help treat hair loss.

c-kit affects hair growth and color in alopecia areata and androgenetic alopecia.

A new gene mutation may allow some piebaldism patients to regain skin color in white patches.

Iron chloride helps create compounds that could be useful in medicine, like treating hair loss.