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Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

LED light therapy at 863 nm wavelength can slow down skin tumor growth and reduce inflammation in mice.

TPA promotes hair growth by increasing stem cell activity and activating specific cell signals.

Hair regrowth slows with age and can be affected by treatments that change enzyme activity in the skin.

The research found ways to activate melanocyte stem cells for potential treatment of skin depigmentation conditions.

Blocking mTORC1 reduces skin tumor growth in mice.

IL-1β inhibits human hair follicle growth.

ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

Alpha-difluoromethylornithine prevents cancer in mice but causes hair loss.

Active oxygen species might be involved in skin tumor growth, but their exact role is unclear.

Activated ras can protect kidney cells from a certain substance that causes cell death.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

The model can effectively test gene functions and drug responses in human skin.

Mirex seems to promote a unique group of skin cells different from those affected by another tumor promoter, TPA.

Indian Gooseberry has potential for cancer prevention and treatment and promotes hair growth.

Understanding where cancer cells come from helps create better prevention and treatment methods.

Keratin proteins are crucial for healthy skin, but mutations can cause skin disorders with no effective treatments yet.

The article concludes that hair loss is a common side effect of drugs treating skin cancer by blocking the hedgehog pathway, but treatment should continue, and more selective drugs might prevent this side effect.

Removing integrin α3β1 from hair stem cells lowers skin tumor growth by affecting CCN2 protein levels.

Meis1 is crucial for skin health and tumor development.

A small group of slow-growing cells causes basal cell carcinoma to return after treatment.

Blocking COX, especially COX-2, in the skin can reduce inflammation and pain and may help prevent skin cancer.

Stem cells, especially from fat tissue and Wharton's jelly, can potentially regenerate hair follicles and treat hair loss, but more research is needed to perfect the treatment.

Wnt and Shh signaling are key in noggin-induced tumors, and blocking them can slow tumor growth.

Blocking mTORC1 may help prevent skin cancer by stopping the growth of certain skin stem cells.

Rapamycin reduces skin cell growth and tumor development by affecting cell signaling in mice.

mrp/plf-mRNA can indicate tumor-promoting effects in skin.

Claudin expression changes help the skin respond to injury.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

PKC ε increases hair follicle stem cell turnover and may raise skin cancer risk.