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Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Modulating BMP activity changes the number, size, shape, and type of ectodermal organs.

The skin's internal clock affects healing, cancer risk, aging, immunity, and hair growth, and disruptions can harm skin health.

Macrophages are vital for skin healing, hair growth, salt balance, and cancer defense.

Ancient Chinese goats evolved cashmere-producing traits due to selective breeding, particularly in genes affecting hair growth.

The article concludes that hair loss is a common side effect of drugs treating skin cancer by blocking the hedgehog pathway, but treatment should continue, and more selective drugs might prevent this side effect.

A specific microRNA, chi-miR-30b-5p, slows down the growth of hair-related cells by affecting the CaMKIIδ gene in cashmere goats.

Scientists created a new cell line from Cashmere goat hair and found that cytokeratin 13 is a unique marker for certain skin cells.

Hair follicle development in cashmere goats involves dynamic changes in proteins and metabolites, with key roles for oxytocin, MAPK, and Ca2+ pathways.

Advancements in tissue engineering show promise for hair follicle regeneration to treat hair loss.

Hair growth phase and certain genes can speed up wound healing, while an inflammatory mediator can slow down new hair growth after a wound. Understanding these factors can improve tissue regeneration during wound healing.

Certain 4-azasteroids are effective at blocking the enzyme that processes testosterone in human skin and could help treat acne, excessive hair growth, and male pattern baldness.

Foxi3 expression in developing teeth and hair is controlled by the ectodysplasin pathway.

Alopecia areata is a hair loss condition caused by immune factors and can be treated with JAK inhibitors.

Certain long non-coding RNAs in cashmere goats affect hair growth when treated with a specific growth factor.

A20 protein is crucial for normal skin and hair development.

Alopecia areata causes hair loss due to an immune attack on hair follicles, influenced by genetics and environment.

Dermal EZH2 controls skin cell growth and differentiation in mice.

Estrogen can temporarily slow down hair growth but this can be reversed.

Changthangi goats have specific genes that help produce Pashmina wool.

Researchers used CRISPR/Cas9 to create a goat with a gene that increased cashmere production by 74.5% without affecting quality.

Melatonin affects cashmere growth in goats by influencing stem cell and certain signaling pathways.

The research found ways to activate melanocyte stem cells for potential treatment of skin depigmentation conditions.

Ezh2 controls skin development by balancing signals for dermal and epidermal growth.

Double-stranded RNA activates a pathway that causes a skin protein to be expressed in the wrong place.

Certain RNA molecules are important for the development of wool follicles in sheep.

Boosting β-catenin signaling in certain skin cells can enhance hair growth.

Researchers identified specific genes that are important for mouse skin cell development and healing.

Melatonin treatment increases a specific RNA in goat cells that boosts cashmere growth.

Bmpr2 and Acvr2a receptors are crucial for hair retention and color.