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The document concludes that mouse models are helpful but have limitations for skin wound healing research, and suggests using larger animals and genetically modified mice for better human application.

Pangolins have lost some skin-related genes, but kept others, leading to their unique scales and skin features.

Vav2 and Vav3 proteins help control skin stem cell numbers and activity in both healthy and cancerous cells.

The nervous system helps control stem cell behavior and immune responses, affecting tissue repair and maintenance.

Hair follicle bulge cells don't help skin regrow after glucocorticoid damage; interfollicular epidermis cells do.

Skin cells show flexibility in healing wounds and forming tumors, with potential for treating hair disorders and chronic ulcers.

Maintaining DNA integrity in stem cells is crucial to prevent aging and cancer.

Stem cells from hair follicles can safely treat hair loss.

New treatments targeting skin stem cells show promise for skin repair, anti-aging, and cancer therapy.

Ca_v1.2 affects hair growth and could be a target for hair loss treatments.

The conclusion is that skin and hair patterns are formed by a mix of cell activities, molecular signals, and environmental factors.

MicroRNA-148a is crucial for maintaining healthy skin and hair growth by affecting stem cell functions.

Autophagy helps control skin inflammation and cancer responses and regulates hair growth by affecting stem cell activity.

Genetic mutations cause various hair diseases, and whole genome sequencing may reveal more about these conditions.

PDGFA/AKT signaling is important for the growth and maintenance of certain skin fat cells.

The document concludes that identifying the specific cells where skin cancers begin is important for creating better prevention, detection, and treatment methods.

Mice without the Sept4/ARTS gene heal wounds better due to more stem cells that don't die easily.

Scientists have made progress in understanding hair follicle stem cells, identifying specific genes and markers, and suggesting their use in treating hair and skin conditions.

The article concludes that better understanding gene regulation related to seasonal changes can offer insights into the mechanisms of seasonal timing in mammals.

Morroniside may help hair grow and stay in its growth phase by affecting certain cell signals.

GRK2 is essential for healthy hair follicle function, and its absence can lead to hair loss and cysts.

Conditioned media from mesenchymal stem cell cultures could be a more effective alternative for regenerative therapies, but more research is needed.

The document concludes that better understanding the wound microbiome can improve chronic wound care by preserving helpful bacteria and targeting harmful ones.

When skin blisters, healing the wound is more important than growing hair, and certain stem cells mainly fix the blisters without helping hair growth.

The document concludes that understanding the genes and pathways involved in hair growth is crucial for developing treatments for hair diseases.

Skin-derived stem cells can become various cell types, including germ cell-like and oocyte-like cells.

Using developmental signaling pathways could improve adult wound healing by mimicking scarless embryonic healing.

Quercetin significantly helps hair growth by activating hair follicles and improving blood vessel formation around them.

Dermal fibroblasts have adjustable roles in wound healing, with specific cells promoting regeneration or scar formation.

Phospholipase A2 enzymes play key roles in skin health and disease.