Search

everythinglearnresearchcommunity

for

Search:↓

Lymphoid-specific helicase (Lsh) is crucial for skin growth, change, and healing after injury.

Regenerated hairs can regain their color if the wound occurs during a certain stage of hair growth, and this process is helped by specific skin cells and proteins.

Dermal stem cells help regenerate hair follicles and heal skin wounds.

Different cell types work together to repair skin, and targeting them may improve healing and reduce scarring.

After skin is damaged, noncoding dsRNA helps prostaglandins and Wnts work together to repair tissue and promote hair growth.

Wounded skin cells can revert to stem cells and help heal.

Rats can't grow new hair follicles after skin wounds, unlike mice, due to differences in gene expression and response to WNT signaling.

The Ovol2-Zeb1 circuit is crucial for skin healing and hair growth by guiding cell movement and growth.

Wound healing can help prevent hair loss from chemotherapy in young rats by increasing interleukin-1β signaling.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

Estrogen loss during menopause worsens skin health, but hormone replacement therapy may improve it, though more research is needed.

Hair follicles can regrow in wounded adult mouse skin using a process like embryo development.

The biofilm-dispersing wound gel helps wounds heal faster and prevents infection.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

Glycogen levels in mouse skin drop after injury but increase during healing, returning to normal within a month.

Softer hydrogels help wounds heal better with less scarring.

Hair can regrow in large wounds through a process similar to how hair forms in embryos, and understanding this could lead to new treatments for hair loss or scarring.

DPP4, a molecule in skin, helps heal large wounds and regrow hair follicles when its levels are reduced.

Smad2/3-dependent TGF-β signaling increases during wound healing.

The African spiny mouse can fully regenerate its muscle without scarring, unlike the common house mouse.

The African spiny mouse heals skin without scarring due to different protein activity compared to the common house mouse, which heals with scarring.

MRL/MpJ mice's skin wounds heal with scars, unlike their ear wounds which can regenerate.

Fibroblast growth factors are crucial for hair follicle development and regeneration.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

House fly larvae substances improve wound healing and skin regeneration, especially in immunosuppressed mice.

Epigenetic changes are crucial for stem cell behavior in skin wound healing and their disruption may lead to cancer.

Skin bacteria, specifically Streptococcus and Staphylococcus, help in hair regrowth after skin injury and speed up wound healing.

The research found that different types of fibroblasts are involved in wound healing and that some blood cells can turn into fat cells during this process.

Careful selection of mice by genetics and age, and controlled housing conditions improve the reliability of hair regrowth in wound healing tests.

Planarian regeneration begins with a specific gene activation caused by injury, essential for healing and tissue regrowth.