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After skin is damaged, noncoding dsRNA helps prostaglandins and Wnts work together to repair tissue and promote hair growth.

Wounded skin cells can revert to stem cells and help heal.

Lymphoid-specific helicase (Lsh) is crucial for skin growth, change, and healing after injury.

Dermal stem cells help regenerate hair follicles and heal skin wounds.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

DPP4, a molecule in skin, helps heal large wounds and regrow hair follicles when its levels are reduced.

Glycogen levels in mouse skin drop after injury but increase during healing, returning to normal within a month.

The research found that different types of fibroblasts are involved in wound healing and that some blood cells can turn into fat cells during this process.

MRL/MpJ mice's skin wounds heal with scars, unlike their ear wounds which can regenerate.

The African spiny mouse can fully regenerate its muscle without scarring, unlike the common house mouse.

The African spiny mouse heals skin without scarring due to different protein activity compared to the common house mouse, which heals with scarring.

Smad2/3-dependent TGF-β signaling increases during wound healing.

Careful selection of mice by genetics and age, and controlled housing conditions improve the reliability of hair regrowth in wound healing tests.

Epigenetic changes are crucial for stem cell behavior in skin wound healing and their disruption may lead to cancer.

Fibroblast growth factors are crucial for hair follicle development and regeneration.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

Different cell types work together to repair skin, and targeting them may improve healing and reduce scarring.

Hair follicles can regrow in wounded adult mouse skin using a process like embryo development.

Hair can regrow in large wounds through a process similar to how hair forms in embryos, and understanding this could lead to new treatments for hair loss or scarring.

The Ovol2-Zeb1 circuit is crucial for skin healing and hair growth by guiding cell movement and growth.

Planarian regeneration begins with a specific gene activation caused by injury, essential for healing and tissue regrowth.

Damage to skin releases dsRNA, which activates TLR3 and helps in skin and hair follicle regeneration.

Skin and hair can help us understand organ regeneration, especially how certain stem cells might be used to form new organs.

Rats can't grow new hair follicles after skin wounds, unlike mice, due to differences in gene expression and response to WNT signaling.

Wound healing can help prevent hair loss from chemotherapy in young rats by increasing interleukin-1β signaling.

Skin bacteria, specifically Streptococcus and Staphylococcus, help in hair regrowth after skin injury and speed up wound healing.

Thymic stromal lymphopoietin (TSLP) promotes hair growth, especially after skin injury.

Activating the GDNF-GFRα1-RET signaling pathway could potentially promote skin and limb regeneration in humans and could be used to treat hair loss and promote wound healing.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.