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As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Researchers found new potential treatments for conditions related to the androgen receptor, like male hormonal contraception, by testing thousands of compounds.

MR antagonists may improve skin health and wound healing, especially in aging.

Blocking the mineralocorticoid receptor can help treat skin thinning caused by steroids.

A new treatment called SAMiRNA-AR68 increases hair count in people with hair loss, showing similar results to existing treatments but without side effects.

The study found that androgen receptors in skin cells mainly affect the focal adhesion pathway and control the caveolin-1 gene, with implications for new treatments for related diseases.

Understanding how androgens and their receptors work can lead to improved treatments for skin diseases.

A new genetic change causing early stop in the androgen receptor gene was found in a patient with androgen insensitivity syndrome.

New treatments targeting androgen receptors show promise for drug-resistant prostate cancer.

Hairless protein can block vitamin D activation in skin cells.

The document concludes that targeting 5α-reductase, the androgen receptor, and hair growth genes, along with using compounds with anti-androgenic properties, could lead to more effective hair loss treatments.

The conclusion is that genetic changes in the glucocorticoid receptor can lead to conditions affecting stress response, immunity, and metabolism, requiring personalized treatment.

The conclusion suggests that prostate cancer should be classified by castration status and that new therapies targeting androgen receptor signaling show promise.

The vitamin D receptor can work without its usual activating molecule.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

TCDD disrupts skin stem cells, causing skin issues like chloracne.

Certain gene variations in the Vitamin D Receptor are linked to higher risk of female hair loss.

TDM10842, a thyroid hormone receptor activator, was found to effectively promote hair growth in mice.

Fewer GGC sequences in the androgen receptor gene improve finasteride treatment for hair loss.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

A specific gene mutation causes congenital hair loss.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

TLR4 is important in aging-related diseases and could be a new treatment target.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

Vitamin D receptor is crucial for skin wound healing.