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Alopecia areata involves both innate and adaptive immunity, with specific genes linked to the disease.

Porcine skin is very similar to human skin, making it a useful model for research.

Hydrogel scaffolds can help wounds heal better and grow hair.

The skin has a complex immune system that is essential for protection and healing, requiring more research for better wound treatment.

Hair follicles prevent NK cell attacks to avoid hair loss.

The immune system plays a key role in tissue repair, affecting both healing quality and regenerative ability.

Cancer treatments targeting specific cells and the immune system can cause skin, mouth, hair, and nail problems, affecting patients' quality of life and treatment adherence.

Inactive stem cells in hair follicles and muscles can avoid detection by the immune system.

Transplant success has improved with better immunosuppressive drugs and donor matching.

The nail matrix has a reduced immune response, protecting it from autoimmunity.

Masculine facial features may not reliably indicate heritable health, and more research is needed.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

Men generally have more severe COVID-19 cases and higher death rates than women due to biological differences.

Restoring hair bulb immune privilege is crucial for managing alopecia areata.

The study found that two enzymes linked to hair loss are located in different parts of the scalp, supporting a common treatment's effectiveness.

Damaging mutations in NFKB2 cause a severe and distinct form of primary immunodeficiency with early-onset and often ACTH-deficiency.

Targeting TGFβ can improve skin immunity in older people.

Smaller nanoemulsions can penetrate skin and hair follicles better, which may be useful for delivering drugs and vaccines through the skin.

Cancer patients treated with immune checkpoint inhibitors may develop alopecia, but some hair regrowth is possible with treatment.

Some vaccines might rarely cause hair loss, more research is needed.

Mycophenolate mofetil may improve lupus nephritis remission more than cyclophosphamide but with uncertain safety.

Melanocyte activity in hair follicles is linked to the hair growth cycle, being active in growth phases and inactive in rest phases.

Animal models have helped understand hair loss from alopecia areata and find new treatments.

Markers help differentiate between apocrine and eccrine sweat glands to identify sweat gland tumors.

A deficiency in the TTC7A gene causes immune problems, gut issues, and hair loss.

The study found that immune responses disrupt hair growth cycles, causing hair loss in alopecia areata.

Anti-TNF-α therapy may increase the risk of developing alopecia areata, especially in those with a history of autoimmune disease.

Immunosuppressive and anti-TNF therapies in IBD patients can increase the risk of skin cancer and cause various skin issues.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

The document concludes that while there are various treatments for Alopecia Areata, there is no cure, and individualized treatment plans are essential due to varying effectiveness.