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Skin stem cells interacting with their environment is crucial for maintaining and regenerating skin and hair, and understanding this can help develop new treatments for skin and hair disorders.

Lgr5 is a marker for active, self-renewing stem cells in the intestine and skin, important for tissue maintenance.

Super-enhancers controlled by pioneer factors like SOX9 are crucial for stem cell adaptability and identity.

Stem cell offspring help control their parent stem cells, affecting tissue health, healing, and cancer.

Different small areas within hair follicles send specific signals that control what type of cells stem cells become.

Different types of skin cells have unique genetic markers that affect how likely they are to spread cancer.

Inactive stem cells in hair follicles and muscles can avoid detection by the immune system.

Telocytes might help with skin repair and regeneration.

Stem cell self-renewal is complex and needs more research for full understanding.

The WNT signaling pathway is important in many diseases and targeting it could offer new treatments.

Skin aging is due to impaired stem cell mobilization or fewer responsive stem cells.

Different types of stem cells and their environments are key to skin repair and maintenance.

Cytokeratin 19 and cytokeratin 15 are key markers for monitoring the quality and self-renewing potential of engineered skin.

New treatments targeting specific genes show promise for treating keratin disorders.

Skin development in mammals is controlled by key proteins and signals from underlying cells, involving stem cells for maintenance and repair.

Mice without the Sept4/ARTS gene heal wounds better due to more stem cells that don't die easily.

The extracellular matrix is crucial for controlling skin stem cell behavior and health.

Skin stem cells maintain and repair the outer layer of skin, with some types being essential for healing wounds.

MicroRNAs are crucial for controlling skin development and healing by regulating genes.

Mice with human skin protein K8 had more skin problems and cancer.

Using the drugs AMD3100 and Tacrolimus together greatly improves skin healing and hair growth after a deep skin cut by increasing stem cells in the wound.

Stem cells have great potential for improving wound healing, but more research is needed to find the best types and ways to use them.

FA2H is essential for normal fur and sebum production in mice.

GLI2 increases follistatin production in human skin cells.

Transit-amplifying cells are crucial for tissue repair and can contribute to cancer when they malfunction.

Platelet-rich plasma (PRP) is a simple, cost-effective treatment that promotes hair growth and reduces hair loss, with high patient satisfaction.

Using neurohormones to control keratin can lead to new skin disease treatments.

TGM3 is important for skin and hair structure and may help diagnose cancer.

Rats can't grow new hair follicles after skin wounds, unlike mice, due to differences in gene expression and response to WNT signaling.

The research suggests that autophagy-related genes might play a role in causing alopecia areata.