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Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Epithelial stem cells can adapt and help in tissue repair and regeneration.

T cells and bacteria in the gut and skin help maintain health and protect against disease.

Scientists turned rat thymus cells into stem cells that can help repair skin and hair.

Sensory neuron remodeling and Merkel-cell changes in the skin happen independently.

Sensory neuron and Merkel-cell changes in the skin happen independently during normal skin maintenance.

Sensory neuron and Merkel cell changes in the skin happen independently during normal skin maintenance.

Understanding phenotypic plasticity is crucial for developing effective cancer therapies.

Sensory neuron changes and Merkel-cell changes in the skin happen independently during normal skin maintenance.

Sensory neuron remodeling and Merkel-cell changes happen independently during skin maintenance.

Sensory neuron and Merkel cell changes in the skin happen independently during normal skin maintenance.

Sensory neurons and Merkel cells remodel at different rates during normal skin maintenance.

Adult esophageal cells can start to become like skin cells, with a key pathway influencing this change.

Different types of skin cells have unique genetic markers that affect how likely they are to spread cancer.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

Super-enhancers controlled by pioneer factors like SOX9 are crucial for stem cell adaptability and identity.

Activating β-catenin in mammary cells leads to changes that cause early-stage abnormal growths similar to skin structures.

The study showed that hair follicle stem cells can maintain and organize themselves in a lab setting, keeping their ability to renew and form hair and skin.

The Ovol2-Zeb1 circuit is crucial for skin healing and hair growth by guiding cell movement and growth.

Stem cells in the hair follicle are regulated by their surrounding environment, which is important for hair growth.

Different types of skin cells play specific roles in development, healing, and cancer.

The enzymes Tet2 and Tet3 are important for skin cell development and hair growth.

Certain mature cells in mouse lungs can turn back into stem cells to aid in tissue repair.

Different human hair follicle stem cells grow at different rates and respond differently to a baldness-related compound.

Macrophage iron release is crucial for hair growth and wound healing.

Ovol2 is crucial for hair growth and skin healing by controlling cell movement and growth.

TGF-β signaling is crucial for stem cell maintenance, differentiation, and has implications for cancer treatment.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

Nerve signals are crucial for hair follicle stem cells to become skin stem cells and help in wound healing.

Intestinal stem cells can help repair skin damage from radiation.