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Research 30 of 555
- Identification of dkk4 as a target of Eda-A1/Edar pathway reveals an unexpected role of ectodysplasin as inhibitor of Wnt signalling in ectodermal placodes
- Ectodysplasin A (EDA) Signaling: From Skin Appendage to Multiple Diseases
- Interplay between EDA-EDAR and WNT signalling pathways in the development of skin appendages in hypohidrotic ectodermal dysplasia
- Ectodysplasin-A (EDA) Signaling Cross-Talk in Skeletogenesis
- Coordinating the Sculptors: Ectodysplasin-A (EDA) Signaling Cross-Talk with Skeletogenic Pathways
- Involvement of Wnt, Eda and Shh at defined stages of sweat gland development
- Edar Signaling in the Control of Hair Follicle Development
- EDA Missense Variant in a Cat with X-Linked Hypohidrotic Ectodermal Dysplasia
- The role of hair follicles and Edar signalling in cutaneous wound healing
- Feather arrays are patterned by interacting signalling and cell density waves
- Avaliação do perfil fenotípico, proliferativo e secretório de células-tronco mesenquimais do tecido adiposo captadas por lipoaspiração assistida por laser
- Farmácia Padrão da Légua, Custóias e nos Serviços Farmacêuticos da Klinicka Bolnica Dubrava, Croácia
- Keratinocyte-specific ablation of the NF-κB regulatory protein A20 (TNFAIP3) reveals a role in the control of epidermal homeostasis
- Lymphotoxin-β regulates periderm differentiation during embryonic skin development
- 15. Hair and Nails
- Anatomy and Physiology of Hair
- Biology and Genetics of Hair
- TNF superfamily in skin appendage development
- Dynamic Signals for Hair Follicle Development and Regeneration
- Development and Maintenance of Epidermal Stem Cells in Skin Adnexa
- Hair Follicle Morphogenesis During Embryogenesis, Neogenesis, and Organogenesis
- Lgr5 Stem Cells and Potential Treatments for Hair Loss
- Ectodysplasin-A2 induces dickkopf 1 expression in human balding dermal papilla cells overexpressing the ectodysplasin A2 receptor
- Current Genetics in Hair Diseases
- Skin‐on‐a‐chip strategies for human hair follicle regeneration
- A new mutation resulting in the truncation of the TRAF6-interacting domain of XEDAR: a possible novel cause of hypohidrotic ectodermal dysplasia: Figure 1
- EDA Fibronectin Microarchitecture and YAP Translocation During Wound Closure
- Ectodysplasin Signaling in Cutaneous Appendage Development: Dose, Duration, and Diversity
- Identification of Ectodysplasin Target Genes Reveals the Involvement of Chemokines in Hair Development
- Edar/Eda interactions regulate enamel knot formation in tooth morphogenesis