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Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

The document concludes that the development of certain tumors is influenced by genetic background and that a specific gene modification can lead to tumor regression and reduced growth.

Researchers found specific genes in the part of hair follicles that could help treat hair disorders.

Noggin gene inactivation causes skeletal defects in mice, varying by genetic background.

Too much Smad7 can cause serious changes in skin tissues, including problems with hair growth, thymus shrinkage, and eye development issues.

The study concluded that a protein important for hair strength is regulated by certain molecular processes and is affected by growth phases.

The protein STAT3 slows down cell growth by blocking the FST gene, which affects hair development in sheep.

Melatonin helps goat hair stem cells grow and maintain their ability to become different cell types.

Fibroblast Growth Factors (FGFs) are important for tissue repair and regeneration, influencing cell behavior and other factors involved in healing, and are crucial in processes like wound healing, bone repair, and hair growth.

Smad-4 and Smad-7 are key in hair follicle development, with other Smads being less important.

Dermal fibroblasts have adjustable roles in wound healing, with specific cells promoting regeneration or scar formation.

Growth factors are crucial for hair development and could help treat hair diseases.

Epithelial-mesenchymal interactions control hair growth cycles through specific molecular signals.

Dermal papilla cells can help regrow hair and are promising for hair loss treatments.

Nanocarriers with plant extracts show promise for safe and effective hair growth treatment.

Sweat glands and hair follicles are determined by opposing signals, with BMPs promoting sweat glands and blocking BMPs leading to hair follicles.

BMP signaling controls hair follicle size and cell growth by affecting cell cycle genes.

BMPR1a-ECD reduces wrinkles much more effectively than retinoic acid.

BMP2 and BMPR-IA may stop hair growth while Noggin may encourage it in yak skin.

TLR2 helps control hair growth and regeneration, and its reduction with age or obesity can impair hair growth.

Follistatin helps hair growth and cycling, while activin prevents it.

Disrupted stem cell signals in hairpoor mice cause hair loss.

The conclusion is that Fgf18 and Tgf-ß signaling could be targeted for hair loss treatments.

Understanding hair follicle development can help treat hair loss, skin regeneration, and certain skin cancers.

The conclusion is that skin and hair patterns are formed by a mix of cell activities, molecular signals, and environmental factors.

Blocking BMP signaling causes hair loss and disrupts hair growth cycles.

Alkylation of human hair keratin allows for adjustable drug release rates in hydrogels for medical use.

Different stem cells are key for hair growth and health, and understanding their regulation could help treat hair loss.

Hair growth and development are controlled by specific signaling pathways.

Fibroblasts are crucial for tissue repair and inflammation, and understanding them can help treat fibrotic diseases.