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Personalized medicine in dermatology uses molecular biomarkers to improve diagnosis and treatment but needs further advancements for practical use.

Tissue stiffness affects hair follicle regeneration, and Twist1 is a key regulator.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

Integrin alphavbeta6 is important for wound healing and hair growth, and blocking it may improve these processes.

The skin's internal clock affects healing, cancer risk, aging, immunity, and hair growth, and disruptions can harm skin health.

Further research is needed to improve hair regeneration using stem cells and nanomaterials.

Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

Prostaglandin D2 increases testosterone levels in skin cells through reactive oxygen species, not enzymes, which could lead to new hair loss treatments.

A new hydrogel made from human cells improves wound healing by working with immune cells to promote repair.

Certain signals and genes play a key role in hair growth and regeneration, and understanding these could lead to new treatments for skin regeneration.

Androgen receptor activity blocks Wnt/β-catenin signaling, affecting hair growth and skin cell balance.

Aging in hair follicle stem cells leads to hair graying, thinning, and loss.

Stem cells help heal skin wounds by moving and changing roles, working with other cells, and needing more research on their activation and behavior.

Activating the Wnt/β-catenin pathway could lead to new hair loss treatments.

Damage to skin releases dsRNA, which activates TLR3 and helps in skin and hair follicle regeneration.

The research found that different types of fibroblasts are involved in wound healing and that some blood cells can turn into fat cells during this process.

Understanding how baby skin heals without scars could help develop treatments for adults to heal wounds without leaving scars.

Different immune cells like platelets, mast cells, neutrophils, macrophages, T cells, B cells, and innate lymphoid cells all play roles in skin wound healing, but more research is needed due to inconsistent results and the complex nature of the immune response.

Activating a protein called β-catenin in adult skin can make it behave like young skin, potentially helping with skin aging and hair loss.

The African spiny mouse heals skin without scarring due to different protein activity compared to the common house mouse, which heals with scarring.

Fat cells in the skin help start healing and form important repair cells after injury.

Low-energy fractional CO2 laser therapy significantly improves hair regrowth in women with female-pattern hair loss.

The book "Hair Follicle Regeneration" discusses the potential of regenerating human hair follicles or activating dormant ones as a possible cure for baldness, and the promising role of new technologies like 3D printing in this field.

M2 macrophages help hair regrowth in wounds by making growth factors.

CyRL-QN15 peptide boosts hair growth in diabetic mice by activating specific cell pathways.

Wnt signaling is crucial for skin and hair development and its disruption can cause skin tumors.

Different fibroblasts play key roles in skin healing and scarring.

Activating TRPV4 in skin cells helps regrow hair in mice, possibly offering a treatment for hair loss.

Wnt signaling controls whether hair follicle stem cells stay inactive or regenerate hair.

Engineered skin substitutes can grow hair but have limitations like missing sebaceous glands and hair not breaking through the skin naturally.