Search

everythinglearnresearchcommunity

for

Search:↓

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

The transgene likely activated an oncogene or interrupted a tumor suppressor gene, causing melanoma in mice.

Skin tumor regression is helped by retinoic acid signaling blocking Wnt signaling.

Activin increases skin tumor formation, skin Tregs help hair growth, lymph-node removal doesn't improve melanoma survival, cells can revert to stem cells in wound healing, and skin bacteria produce peptides that may treat infections.

The symposium showed that stem cells are key for understanding and treating skin diseases and for developing new skin models and therapies.

Organoids created from stem cells are used to model diseases, test drugs, and develop personalized and regenerative medicine.

New cancer treatments show promise in reducing tumor growth and improving skin regeneration in mice.

Stem cells are key for the growth, upkeep, and repair of sebaceous glands and for understanding skin disorders.

In vivo lineage labelling is better than in vitro methods for identifying and understanding stem cells.

Some skin tumors may start from hair follicle stem cells.

Scientists now better understand how human hair growth is controlled, including the roles of specific genes and proteins.

The document concludes that while some organisms can regenerate body parts, mammals generally cannot, and cancer progression is complex, involving mutations rather than a strict stem cell hierarchy.

The author felt excited and honored to receive the 2016 Early Career Life Scientist Award.

Neoplasms hide in hair follicles to avoid the immune system.

The study concludes that the EDA2R gene is activated by p53 during chemotherapy but is not necessary for chemotherapy-induced hair loss.

Blocking YAP/TAZ could be a new way to treat skin cancer.

Mutations in the SHH pathway in certain skin cells can cause skin tumors and abnormal hair growth.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

Polyomavirus A2 infection in newborn mice caused hair follicle tumors.

A small group of slow-growing cells causes basal cell carcinoma to return after treatment.

Mice studies show that Protein Phosphatase 2A is crucial for cell growth, development, and disease prevention.

New findings suggest potential treatments for melanoma, hyperpigmentation, hair defects, and multiple sclerosis, and show skin microbiome changes don't cause atopic dermatitis.

CEC levels may be a useful marker for predicting prostate cancer progression.

Basal cell carcinoma mostly starts from cells in the upper skin layers, not hair follicle stem cells.

Low doses of some substances can be beneficial, while high doses can be harmful or toxic.

New therapies for Birt–Hogg–Dubé syndrome are being developed based on understanding the FLCN gene's role.

Lecithin-encapsulated resveratrol nanoparticles could be a safe and effective anti-cancer treatment.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

Stem cell therapies show promise for treating hair loss, but more research is needed to understand their safety and effectiveness.

GLI1 might protect against the start of skin cancer and is not linked to cancer severity.