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Human dental pulp stem cell-conditioned medium, especially from hypoxic conditions, may help treat chemotherapy-induced hair loss and does not increase cancer risk.

Adipokines affect skin health and could be targeted for treating skin diseases.

Exosome treatment for hair growth is promising but not FDA-approved and needs more research on safety and how it works.

Early diagnosis and treatment of Hidradenitis Suppurativa are important to prevent serious physical and mental effects.

Lifestyle factors like BMI at young adulthood can increase cancer risk in women with Lynch Syndrome, but diet and height don't seem to affect this risk.

Runx1 affects hair growth, cancer development, and autoimmune diseases in epithelial tissues.

The transgene likely activated an oncogene or interrupted a tumor suppressor gene, causing melanoma in mice.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Hair structure worsens as tumors grow in mice.

Rapamycin reduces skin cell growth and tumor development by affecting cell signaling in mice.

Excessive putrescine causes hair loss in transgenic mice by disrupting hair follicle development.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.

Researchers found an RNA transcript that might help control a growth factor linked to tumor development.

Antizyme reduces tumor growth and normalizes skin cell development affected by MEK.

Lacking cyclin D3 reduces skin cancer growth without affecting normal skin cell growth.

Polyomavirus A2 infection in newborn mice caused hair follicle tumors.

Skin health and diseases are closely linked to metabolic processes.

Beta-HPV and MCPyV are linked to certain skin cancers, with ongoing research and vaccine development.

Meis1 is crucial for skin health and tumor development.

Active oxygen species might be involved in skin tumor growth, but their exact role is unclear.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

Ozone therapy might improve cancer treatment and reduce its side effects.

Antizyme slows skin tumor growth by reducing cell growth in mice.

High levels of ornithine decarboxylase can cause tumors in mouse skin.

Skin cells show flexibility in healing wounds and forming tumors, with potential for treating hair disorders and chronic ulcers.

Cells from a skin condition can create new hair follicles and similar growths in mice, and a specific treatment can reduce these effects.

Wnt and Shh signaling are key in noggin-induced tumors, and blocking them can slow tumor growth.