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Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.

Lacking cyclin D3 reduces skin cancer growth without affecting normal skin cell growth.

Ozone therapy might improve cancer treatment and reduce its side effects.

Hair structure worsens as tumors grow in mice.

The transgene likely activated an oncogene or interrupted a tumor suppressor gene, causing melanoma in mice.

Rapamycin reduces skin cell growth and tumor development by affecting cell signaling in mice.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Researchers found an RNA transcript that might help control a growth factor linked to tumor development.

Antizyme reduces tumor growth and normalizes skin cell development affected by MEK.

Meis1 is crucial for skin health and tumor development.

Polyomavirus A2 infection in newborn mice caused hair follicle tumors.

Antizyme slows skin tumor growth by reducing cell growth in mice.

Active oxygen species might be involved in skin tumor growth, but their exact role is unclear.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

LED light therapy at 863 nm wavelength can slow down skin tumor growth and reduce inflammation in mice.

Blocking mTORC1 reduces skin tumor growth in mice.

A transwoman developed a brain tumor after nine years of hormone therapy, suggesting a possible link between the treatment and tumor development.

Wnt7a protein is crucial for development and tissue maintenance and plays varying roles in diseases and potential treatments.

The Wnt signaling pathway is not a major factor in the development of keratoacanthoma, a type of skin tumor.

Blocking mTORC1 may help prevent skin cancer by stopping the growth of certain skin stem cells.

High levels of ornithine decarboxylase can cause tumors in mouse skin.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

The analysis of a large pilomatricoma revealed five distinct areas with different gene activity related to hair growth and tumor development.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

Patients with Bohring-Opitz syndrome and ASXL1 mutations need regular kidney ultrasounds to check for tumors.

Runx1 affects hair growth, cancer development, and autoimmune diseases in epithelial tissues.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Cancer can hijack the body's cell repair system to promote tumor growth, and targeting this process may improve cancer treatments.