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Research 61–90 of 1000+
- Alopecia
- Stem cell dynamics, migration and plasticity during wound healing
- Type 1 interferon signature in the scalp lesions of alopecia areata
- Vasoactive intestinal peptide, whose receptor-mediated signalling may be defective in alopecia areata, provides protection from hair follicle immune privilege collapse
- Immune cell regulation of the hair cycle
- Pro-inflammatory Vδ1+T-cells infiltrates are present in and around the hair bulbs of non-lesional and lesional alopecia areata hair follicles
- The Overexpression of Tβ4 in the Hair Follicle Tissue of Alpas Cashmere Goats Increases Cashmere Yield and Promotes Hair Follicle Development
- Single-cell analysis of temporal immune cell dynamics in alopecia areata reveals a causal role for clonally expanded CD8+ T cells in disease
- Predominant expression of interleukin (IL)‐17 in chronic alopecia areata compared to IFN‐γ in pathogenic Th17 cells, tissue‐resident memory T cells and natural killer cells
- 085 Autoantigen screening in C3H/HeJ mouse model of alopecia areata revealed high antigenicity of melanocyte-associated antigen epitopes
- Resident T cell activation leads to human hair follicle immune privilege loss ex vivo, which is prevented by the DHODH inhibitor farudodstat: relevance for alopecia areata
- Regulatory T cells in skin utilize the Cxcr4-Cxcl12 axis to promote hair follicle regeneration
- 52993 Regulatory γδ T cells protect human scalp hair follicles from alopecia areata in vivo
- 185 Regulatory γδ T cells protect human scalp hair follicles from alopecia areata in vivo
- Transplanted Hair Follicle Stem Cells Migrate to the Penumbra and Express Neural Markers in a Rat Model of Cerebral Ischaemia/Reperfusion
- In vivo transcriptional governance of hair follicle stem cells by canonical Wnt regulators
- Aging in hair follicle stem cells and niche microenvironment
- An Ovol2‐Zeb1 transcriptional circuit regulates epithelial directional migration and proliferation
- Bulge cells of human hair follicles: segregation, cultivation and properties
- Regulatory T cells in skin mediate immune privilege of the hair follicle stem cell niche
- Are hair follicle stem cells promising candidates for wound healing?
- ROR2 regulates self-renewal and maintenance of hair follicle stem cells
- Inhibition of T-cell activity in alopecia areata: recent developments and new directions
- TREMendous Macrophages Inhibit Hair Growth
- The expanding impact of T-regs in the skin
- 051 γδ T cells as novel players in alopecia areata pathobiology: Vδ1 + T lymphocytes may recognize “stressed” hair follicle keratinocytes, leading to IFNγ-dependent hair follicle dystrophy and immune privilege collapse
- Image 1_Resident T cell activation leads to human hair follicle immune privilege loss ex vivo, which is prevented by the DHODH inhibitor farudodstat: relevance for alopecia areata.pdf
- 0009 Regulatory γδ T cells protect human scalp hair follicles from alopecia areata in vivo and represent potential therapeutic target
- 006 Exogenous IL-27 promotes immunosuppressive T cells and prevents the development of alopecia areata in C3H/HeJ mice
- 1315 An Integrated model of alopecia areata biomarkers highlights both Th1/Th2 upregulation, with stronger correlations between Th2 activation and disease severity