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Vav2 changes how hair follicle stem cells' genes work as they age, which might improve regeneration but also raise cancer risk.

mTOR signaling helps activate hair stem cells by balancing out the suppression caused by BMP during hair growth.

The document concludes that the immune-inhibitory environment of the hair follicle may prevent melanoma development.

Blocking the CCR5 receptor may be a new way to treat hair loss from alopecia areata.

DHT reduces a cell's ability to promote hair growth, while 3D culture without DHT improves it.

The protein aPKCλ is crucial for keeping hair follicle stem cells inactive and for hair growth and regeneration.

Hair loss and aging are caused by the breakdown of a key protein in hair stem cells.

Stem cells help heal skin wounds by moving and changing roles, working with other cells, and needing more research on their activation and behavior.

Alopecia areata shows a unique type 1 interferon signature, suggesting potential treatment by targeting this pathway.

A substance called VIP might protect hair follicles from being attacked by the immune system, and problems with VIP signaling could lead to hair loss in alopecia areata.

Immune cells affect hair growth and could lead to new hair loss treatments.

Rats can't grow new hair follicles after skin wounds, unlike mice, due to differences in gene expression and response to WNT signaling.

Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

Alopecia areata is an autoimmune condition causing hair loss, linked to genetic factors and immune system issues, with no cure yet.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Wnt signaling controls whether hair follicle stem cells stay inactive or regenerate hair.

PEGL-DOX causes Hand-Foot Syndrome due to skin reactions from prolonged circulation and ROS generation.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

miR-218-5p helps skin and hair growth by targeting SFRP2 and activating a specific signaling pathway.

Mice can grow new hair follicles after skin wounds through a process not involving existing hair stem cells, but requiring more research to understand fully.

The document concludes that both internal stem cell factors and external influences like the environment and hormones affect hair loss and aging, with potential treatments focusing on these areas.

Aging in hair follicle stem cells leads to hair graying, thinning, and loss.

The Ovol2-Zeb1 circuit is crucial for skin healing and hair growth by guiding cell movement and growth.

Researchers found that bulge cells from human hair can grow quickly in culture and have properties of hair follicle stem cells, which could be useful for skin treatments.

Hair follicle stem cells are promising for wound healing but require more research for safe clinical use.

Runx genes are important for stem cell regulation and their roles in aging and disease need more research.

Increasing Treg cells in the skin does not cure hair loss from alopecia areata in mice.

Crescina® lotion helps reduce hair loss and increase hair growth in males with androgenetic alopecia.

Restoring immune privilege in hair follicles could help treat certain types of hair loss.

Certain immune cells in the skin can stop hair from growing.