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All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Brain hormones significantly affect hair color and could potentially be used to prevent or reverse grey hair.

Maintaining the right amount of retinoic acid is crucial for healthy hair and skin.

The hair follicle infundibulum plays a key role in skin health and disease, and understanding it better could lead to new skin disease treatments.

Gata6 is important for protecting hair growth cells from DNA damage and keeping normal hair growth.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

TAF4 is important for skin cell growth and helps prevent skin cancer in mice.

OCIAD2 and DCN genes affect hair growth in goats by having opposite effects on a growth signaling pathway and inhibiting each other.

CD201+ fascia progenitors are essential for wound healing and could be targeted for treating skin conditions.

The unique coat of lykoi cats is likely caused by new variants in the Hairless gene.

Gray hair can potentially be reversed, leading to new treatments.

Some vitamin D analogs can thicken skin and reduce pore size like a common acne treatment, with one analog also affecting skin growth factors.

GRK2 is essential for healthy hair follicle function, and its absence can lead to hair loss and cysts.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

ATP-dependent chromatin remodeling is crucial for skin development and stem cell function.

Proretinal nanoparticles are a safe and effective way to deliver retinal to the skin.

CK 5/6 expression in breast cancer is linked to negative hormone receptor status and higher tumor grade.

Chemotherapy significantly lowers Inhibin A levels in breast cancer patients.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

Thyroid hormone receptors are essential for hair growth and wound healing.

Genetic changes in mice help understand skin and hair disorders, aiding treatment development for acne and hair loss.

Hair follicle stem cells can become neural cells using different methods, with varying efficiency.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.