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Finasteride-treated male rats' offspring had altered glucose metabolism, potentially increasing diabetes risk.

Male pattern baldness involves hormone-related hair thinning, shorter hair, and inflammation.

Male and female bodies respond differently to stress, influenced by hormones and development stages, with implications for stress-related diseases.

The document discussed hair loss causes and treatments but didn't give a final summary.

Blocking the androgen receptor in skin cells reduces their growth response to male hormones, suggesting a possible treatment for skin conditions linked to androgens.

The protein ARA70/ELE1 is involved in male pattern baldness, and lower levels of its short form may lead to hair thinning.

Researchers found a gene in hamsters that responds to male hormones and may be indirectly controlled by them.

Targeting androgen receptors could be a promising way to treat skin disorders with fewer side effects.

MC4R gene variants not linked to female hair loss.

Exposure to too much androgen before birth might cause polycystic ovary syndrome later in life.

Lowering testosterone speeds up wound healing in male mice.

Dutasteride and finasteride affect different cell types differently.

A protein called CBP is found in prostate cancer and can increase the effectiveness of certain prostate cancer treatments.

The Androgen Receptor could be a target for treating diseases like cancer, but more research is needed to confirm the effectiveness of potential treatments.

Finasteride, a hair loss drug, may cause long-term sexual side effects due to changes in hormone receptor levels.

KGF and its receptor are found in enlarged prostate tissue and KGF strongly increases cell growth.

Male pattern baldness involves hormones and cell signals affecting hair growth.

The gene SDR5A1 is found in scalp hair of both hirsute and normal individuals, but it does not explain differences in hair growth.

Excess androgens may cause PCOS, not just be a symptom.

Certain gene variations are linked to a higher risk of severe acne, suggesting a genetic influence on the condition.

The androgen receptor is a promising target for breast cancer treatment, especially in triple-negative cases, but more research is needed for personalized therapies.

The balance between androgen receptor and p53 is crucial for sebaceous gland differentiation.

Cells from balding scalps have more androgen receptors than cells from non-balding scalps.

High aldosterone and free testosterone levels link to female hair loss; testing aldosterone may predict hypertension risk.

Dihydrotestosterone changes some hormone-related gene expressions in rat pituitary glands but doesn't affect the estrous cycle.

The conclusion is that genetic changes in the glucocorticoid receptor can lead to conditions affecting stress response, immunity, and metabolism, requiring personalized treatment.

Certain genetic variations are linked to increased androgen levels in PCOS, but more research is needed to understand these connections fully.

Male pattern baldness may be caused by scalp pressure on hair follicles, which increases with age and leads to a cycle of hair loss. This process is not directly determined by genes.

Insulin resistance is a key factor in polycystic ovary syndrome, but genetics may also contribute.

Hormonal interactions, especially involving DHT and estrogen, play a key role in BPH development and treatment.