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NIR-II imaging effectively tracked stem cells that helped repair facial nerve defects in rats.

Using gelatin sponges for deep skin wounds helps bone marrow cells repair tissue without scarring.

New findings suggest potential treatments for melanoma, hyperpigmentation, hair defects, and multiple sclerosis, and show skin microbiome changes don't cause atopic dermatitis.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

Hair follicles prevent NK cell attacks to avoid hair loss.

Cancer treatments targeting specific cells and the immune system can cause skin, mouth, hair, and nail problems, affecting patients' quality of life and treatment adherence.

Accurate diagnosis is key for treating different kinds of hair loss, and immune response variations may affect the condition and treatment results.

People with atopic dermatitis are more likely to develop other skin conditions due to shared genetics and immune pathways.

The new method using gene-modified stem cells and a 3D printed scaffold improved skin repair in mice.

Aged individuals heal wounds less effectively due to specific immune cell issues.

Hydrogel scaffolds can help wounds heal better and grow hair.

The immune system plays a key role in tissue repair, affecting both healing quality and regenerative ability.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Alopecia areata is an autoimmune condition causing hair loss due to the immune system attacking hair follicles, often influenced by genetics and stress.

Alopecia areata involves immune activation in the scalp, suggesting treatments targeting TH1, TH2, and IL-23 pathways.

The nail matrix has a reduced immune response, protecting it from autoimmunity.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

Skin grafts on mice can cause an immune response leading to hair loss, useful for studying human hair loss conditions.

Foxp3 is crucial for regulatory T cell function, and targeting these cells may help treat immune disorders.

Skin's Regulatory T cells are crucial for maintaining skin health and could be targeted to treat immune-related skin diseases and cancer.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

People with late-stage HIV-1 often experience a specific type of hair loss linked to multiple factors, including nutritional issues and immune responses.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

New treatments for immune disorders caused by FOXN1 deficiency are promising.

Hair loss involves immune responses, inflammation, and disrupted signaling pathways.

Alopecia areata causes hair loss due to an immune attack on hair follicles, influenced by genetics and environment.

A boy's growth and immune problems were caused by a new mutation in the STAT5B gene.

Targeting immune tolerance issues in Alopecia Areata could restore hair growth and maintain remission.