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N6-methyladenosine affects hair follicle development differently in Rex and Hycole rabbits.

A synthetic octapeptide may help promote hair growth and counteract hair loss.

Rare genetic variants in five specific genes are linked to male-pattern hair loss but only account for a small part of the risk.

Researchers made a mouse model with curly hair and hair loss by editing a gene.

Protein tyrosine kinases are key in male pattern baldness, affecting skin structure, hair growth, and immune responses.

Genes related to calcium signaling and lipid metabolism are important for curly hair in Mangalitza pigs.

Six genetic conditions are often linked to complete scalp hair loss in children.

Hairlessness in mammals is due to complex genetic changes in both genes and regulatory regions.

Toothpaste containing α-tocopherol acetate, l-menthol, and stevioside can promote hair growth in mice.

SFRP2 and PTGDS may be key factors in female hair loss.

The research suggests that SFRP2 and PTGDS proteins might be indicators of female pattern hair loss and could contribute to hair loss.

Fetal wounds heal without scarring because of different biological factors, which could help improve adult wound healing.

Hair follicle biology advancements may lead to better hair growth disorder treatments.

Hair follicle stem cells use specific chromatin changes to control their growth and differentiation.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

Turing patterns are now recognized as important in developmental biology.

Fetal skin heals without scarring due to unique cells and processes not present in adult skin healing.

The 190-kbp domain contains all human type I hair keratin genes, showing their organization and evolution.

Hair keratin-associated proteins are essential for strong hair, with over 80 genes showing specific patterns and variations among people.

Mice healed without scars as fetuses but developed scars as adults, suggesting scarless healing might be replicated with further research.

Wounds heal without scarring in early development but later result in scars, and studying Wnt signaling could help control scarring.

NcoA4 may have roles beyond helping control gene activity, possibly affecting cell behavior and stability.

Genes linked to fibrosis are more active in people with central centrifugal cicatricial alopecia.

UVB radiation changes the levels of certain microRNAs in skin cells, which may affect cell survival and hair growth.

The study found that expanded skin regenerates similarly to normal skin, with 77 genes playing a role in the process.

Deleting the Far2 gene in mice causes sebaceous gland issues and patchy hair loss.

Troxerutin helps protect skin cells from oxidative stress and may be good for treating hair loss.

Targeting specific miRNAs may help treat hair follicle issues caused by hydrogen peroxide.

A new mutation in the hairless gene causes hair loss and skin wrinkling in mice.

Using skin stem cells and certain molecules might lead to scar-free skin healing.