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Targeting specific T cells may help treat alopecia areata.

IL-15 promotes hair growth and protects hair follicles.

IL-15 helps maintain hair growth and protects the immune status of hair follicles.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

Restoring immune privilege in hair follicles could help treat certain types of hair loss.

Interleukin-15 can help hair growth and protect hair follicles.

A substance called VIP might protect hair follicles from being attacked by the immune system, and problems with VIP signaling could lead to hair loss in alopecia areata.

The document concludes that the immune-inhibitory environment of the hair follicle may prevent melanoma development.

Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

Electrical epilation damages hair follicles and surrounding skin, likely preventing hair regrowth.

Innate lymphoid cells type 1 may contribute to alopecia areata by damaging hair follicles.

Blocking TYK2 might be a new way to treat hair loss from alopecia areata.

Human dermal γδT-cells respond to stress in hair follicles, contributing to hair loss.

ILC1 cells contribute to hair loss in alopecia areata.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

Restoring hair bulb immune privilege is crucial for managing alopecia areata.

IL-15 helps protect hair follicles from immune attacks and encourages hair growth.

Mitochondrial dysfunction may contribute to chronic inflammation and immune system issues in Lichen planopilaris.

Ruxolitinib may help treat hair loss by reducing inflammation, promoting hair growth signals, and protecting hair follicle immunity.

Alopecia areata may be treated by restoring hair follicle immune privilege and adjusting immune responses.

Stress may trigger hair loss by affecting immune protection in hair follicles.

Dandruff is linked to increased T cells and weakened immune protection in hair follicles.

Chronic inflammatory skin diseases are caused by disrupted interactions between skin cells and immune cells.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Immune cells around hair follicles help control hair growth and could be targets for treating hair disorders.

Alopecia areata is an autoimmune condition causing hair loss, linked to genetic factors and immune system issues, with no cure yet.

Alopecia areata is caused by immune system attacks on hair follicles, often triggered by viral infections.

Brain hormones significantly affect hair color and could potentially be used to prevent or reverse grey hair.

Stress-related substance P may lead to hair loss and negatively affect hair growth.

The document concludes that more research is needed to better understand and treat primary cicatricial alopecias, and suggests a possible reclassification based on molecular pathways.