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TRPS1 loss in balding scalp areas contributes to hair loss in androgenic alopecia.

Testosterone can slow hair growth in adult monkeys, but a blocker called RU 58841 can counteract this and potentially help hair regrow.

Testosterone can slow down hair growth when combined with certain cells from bald scalps, and this effect can be blocked by an androgen receptor blocker.

Androgens may cause hair loss by increasing TGF-beta1 from scalp cells, which inhibits hair cell growth.

TGF-β1 from dermal papilla cells suppresses hair growth, and targeting it may help treat androgenetic alopecia.

Male pattern baldness involves hormones and cell signals affecting hair growth.

Dermal Papilla cells are a promising tool for evaluating hair growth treatments.

Androgens increase a growth factor in hair cells by creating reactive oxygen species, and antioxidants might help treat hair loss.

Beard and scalp hair cells have different gene expressions, which may affect beard growth characteristics.

Dihydrotestosterone treatment on 2D and 3D-cultured skin cells slows down hair growth by affecting certain genes and could be a potential target for hair loss treatment.

Testosterone may slow down wound healing and increase inflammation.

Glucocorticoids and sex hormones affect skin health, with potential for targeted treatments to minimize side effects and treat skin conditions.

Male hormones can cause hair loss, but treatments like Minoxidil and Finasteride can help, and targeting TGF-B1 could be a future solution.

Male pattern baldness is caused by certain cells in hair follicles and could potentially be treated by targeting a specific growth factor, TGF-β1.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Androgen receptor signaling causes early aging of cells important for hair growth by damaging their DNA.

The enzyme that changes testosterone to a stronger form is mostly found in the part of the hair follicle called the dermal papilla.

Scientists created cell lines from balding patients and found that cells from the front of the scalp are more affected by hormones that cause hair loss than those from the back.

The gene LRRC15 is more active in balding areas of the scalp compared to non-balding areas.

The research found genes that may protect certain scalp cells from hair loss.

Women with hair loss have more androgen receptors in certain hair follicles.

Inhibiting Zyxin may help treat androgenetic alopecia by promoting hair growth.

Myotonic Dystrophy may age cells faster, and drugs that target aging could be potential treatments.

Hair follicles in androgenetic alopecia age faster, especially in the front.

Changing hair follicle identity could potentially reverse balding.

Androgenetic alopecia, or hair loss, is caused by genetic factors and hormones, with different mechanisms in men and women, and can be linked to insulin resistance and polycystic ovary syndrome.

High levels of testosterone and 5α-DHT can lead to cell death in cells important for hair growth.

PGD2 increases androgen receptor activity in hair cells, which could be targeted to treat hair loss.

Removing STAT5 from 3D-cultured human skin cells reduces their ability to grow hair.