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Chick embryo extract helps rat hair follicle stem cells potentially turn into Schwann cells, important for the nervous system.

Smad1 and Smad5 are essential for hair follicle development and stem cell sleepiness.

SDF-1/CXCL12 and its receptor CXCR4 are crucial for melanocyte movement in mouse hair follicles.

UV exposure reduces Lgr6+ stem cells in mouse skin and they don't significantly contribute to skin cancer development.

Innate lymphoid cells help control skin bacteria by regulating sebaceous glands.

P-cadherin is important for hair growth and health, and its problems can cause hair and skin disorders.

Melanoma's complexity requires personalized treatments due to key genetic mutations and tumor-initiating cells.

Mice lacking a key DNA methylation enzyme in skin cells have a lower chance of activating stem cells necessary for hair growth, leading to progressive hair loss.

MicroRNA-148a is crucial for maintaining healthy skin and hair growth by affecting stem cell functions.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

Different types of resting melanocyte stem cells have unique characteristics and vary in their potential to become other cells.

The we/we wal/wal mice have defects in hair growth and skin layer formation, causing hair loss, useful for understanding alopecia.

Eating a high-fat fish oil diet caused mice to lose hair due to a specific immune cell activity in the skin linked to a protein called E-FABP.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

ILK and ELMO2 help cells move and stick together, important for wound healing and hair growth.

Treatment with plasma rich in growth factors improved hair density and thickness for hair loss patients.

Activating Nrf2 in skin cells speeds up wound healing by increasing the growth of certain stem cells.

P-cadherin is crucial for hair follicle pigmentation but not skin pigmentation.

New single-cell analysis techniques are improving our understanding of stem cells and could help in treating diseases.

Lichen Planopilaris and Frontal Fibrosing Alopecia may help us understand hair follicle stem cell disorders and suggest new treatments.

Different types of inactive melanocyte stem cells exist with unique characteristics and potential to develop into other cells.

The gene Tfap2b is essential for creating a type of stem cell in zebrafish that can become different pigment cells.

Lactate production is important for activating hair growth stem cells.

IL-36α helps grow new hair follicles and speeds up wound healing.

Controlled light treatment in mouse skin speeds up healing and hair growth.

Disrupted cholesterol production impairs hair follicle stem cells, leading to hair loss.

Mice without the Sept4/ARTS gene heal wounds better due to more stem cells that don't die easily.

SCF and c-Kit decrease in AGA hair follicles, possibly affecting hair pigmentation and growth.

Blocking both main energy pathways can stop hair follicle stem cell-induced skin cancer growth.

The secretome from mesenchymal stem cells is a promising treatment that may repair tissue and avoid side effects of stem cell transplantation.