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Trichohyalin is also found in the outer layers of normal human skin.

Sebaceous glands play a key role in skin health, immunity, and various skin diseases.

Targeting NETs may help reduce fibrosis in Crohn's disease.

WNT signaling is crucial for skin development and healing.

The conclusion is that the nuclear lamina and LINC complex in skin cells respond to mechanical signals, affecting gene expression and cell differentiation, which is important for skin health and can impact skin diseases.

Signals from skin cells controlled by Rac proteins help turn certain precursor cells into white fat cells.

Ezh2 controls skin development by balancing signals for dermal and epidermal growth.

Androgens block hair growth by disrupting cell signals; targeting GSK-3 may help treat hair loss.

Androgens prevent hair growth by changing Wnt signals in cells.

Telomere damage affects skin and hair follicle stem cells by messing up important growth signals.

Metabolic signals and cell shape influence how cells develop and change.

FGFs-4, -8, and -9 have overlapping roles and are repeatedly used in tooth development.

Skin development in mammals is controlled by key proteins and signals from underlying cells, involving stem cells for maintenance and repair.

Intu gene is crucial for hair follicle formation by helping keratinocytes differentiate through primary cilia.

NCSTN gene mutation causes abnormal skin cell differentiation and more inflammation, contributing to Hidradenitis Suppurativa.

The right cells and signals can potentially lead to scarless wound healing, with a mix of natural and external wound healing controllers possibly being the best way to achieve this.

Temporary ROS production in cultured human hair follicles promotes growth and stem cell activation.

Stem cell niches are crucial for regulating stem cell renewal and differentiation, and understanding them can help in developing regenerative therapies.

Different small areas within hair follicles send specific signals that control what type of cells stem cells become.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

Scientists improved how to make skin-like structures from stem cells using special gels and a device that controls growth signals, leading to better hair and skin features.

Sweat glands and hair follicles are determined by opposing signals, with BMPs promoting sweat glands and blocking BMPs leading to hair follicles.

Hair follicle development is controlled by interactions between skin tissues and specific molecular signals.

The document concludes that the hair cycle is a complex process involving growth, regression, and rest phases, regulated by various molecular signals.

The document concludes that the hair cycle is a complex process involving growth, regression, and rest phases, regulated by various molecular signals.

The study found that p63 needs signals from morphogens to help skin cells differentiate properly.

The conclusion is that skin and hair patterns are formed by a mix of cell activities, molecular signals, and environmental factors.

After skin injury, adult mice can grow new hair follicles, and this process can be increased or stopped by manipulating Wnt signals.

Different ectodermal organs like hair and feathers regenerate differently, with specific stem cells and signals involved in their growth and response to the environment.

Mutations in Gasdermin A3 cause skin inflammation and hair loss by disrupting mitochondria.