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Dihydrotestosterone (DHT) is crucial for conditions like male-pattern baldness and acne, and measuring a byproduct, androstanediol glucuronide, is a better way to assess DHT's effects than DHT blood levels.

A new method was created to test the effectiveness of Dihydrotestosterone (DHT) inhibitors, like finasteride and dutasteride, in human and fish cells. The results showed fish cells are more sensitive to these treatments, and dutasteride works better than finasteride in all tested cells.

Researchers found four natural compounds that can change DHT levels in prostate cancer cells.

The body's change of testosterone into DHT is not necessary for testosterone's muscle and sexual effects.

Early surgery reduces seizure risk in drug-resistant epilepsy; testosterone to DHT conversion doesn't affect muscle anabolism; veterans with PTSD likely to have riskier opioid use; bevacizumab helps in hereditary bleeding disorder.

Epilation and DHT cause skin calcification by increasing ATP and calcium deposits.

Higher testosterone and DHT levels are linked to hair loss in men, but not to prostate cancer risk.

Vertex baldness has higher DHT levels than frontal baldness, but testosterone and PSA levels are similar.

Male androgenetic alopecia involves hair follicle miniaturization due to DHT, with potential treatments using inhibitors and blockers.

Cedrol nanoemulsions effectively treat hair loss by promoting hair growth and reducing DHT levels safely.

Higher testosterone and DHT levels are not linked to urinary symptoms in men.

Finasteride and dutasteride effectively reduce DHT in hair, which may help evaluate their treatment success for hair loss.

Men with Type 2 Diabetes have lower DHT levels, which may affect fertility and lipid profiles.

Dihydrotestosterone (DHT) increases oil production in skin cells by activating mTOR, and mTOR inhibitors can reduce this effect.

Hormones, especially testosterone and DHT, are key for penis development and function, and testosterone therapy may help with erectile dysfunction in those with low levels.

DHT reduces a cell's ability to promote hair growth, while 3D culture without DHT improves it.

Iron supplements may reverse premature graying in iron-deficient individuals; ingrown nails are common in diabetics with certain risk factors; topical finasteride may reduce scalp DHT as effectively as oral finasteride; monilethrix treatment is challenging but some medications can help.

The sebaceous gland plays a significant role in hair loss and thinning, and using growth stimulators, DHT inhibitors, anti-inflammatory drugs, anti-androgens, and super oxide dismuse substances can help manage it.

Dihydrotestosterone (DHT) and its active metabolites play a key role in various diseases, and the development of 5α-reductase inhibitors can help treat these conditions.

Male pattern baldness is mainly caused by a hormone called dihydrotestosterone (DHT), and drugs that can block this hormone might help prevent hair loss.

Yifa compound effectively treats female pattern hair loss and lowers DHT and DHEA levels.

Combining DHT and EDC improves the strength and stability of PADM scaffolds for tissue engineering.

Cinnamaldehyde helps bone healing initially but may slow healing later unless combined with DHT treatment.

Finasteride safely and effectively treats male pattern hair loss, but may cause reversible sexual issues and harm male fetuses.

The document concludes that understanding hair follicle biology can lead to better hair loss treatments.

Androgens can both increase and decrease hair growth in different parts of the body.

Dutasteride and finasteride may reduce sperm count and volume but don't affect movement or shape; effects are reversible after stopping.

High levels of 3 alpha-diol glucuronide in the blood are a marker of increased androgen action in women with excessive hair growth of unknown cause.

Finasteride effectively blocks enzyme causing male pattern baldness.

Inflammation may be linked to hair loss, and targeting specific enzymes could help treat it.