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Pediatric alopecia areata is more immune-active than adult cases, suggesting age-specific treatments and potential use of JAK inhibitors.

Alopecia areata and vitiligo share immune system dysfunction but differ in specific immune responses and affected areas.

Alopecia areata is an autoimmune condition causing hair loss, influenced by genetics, stress, and diet, and may be prevented by a high soy oil diet.

Minoxidil can cause a skin reaction called ALEP.

Alopecia areata involves both innate and adaptive immunity, with specific genes linked to the disease.

The MAGE3 hypothesis for alopecia areata did not lead to a significant breakthrough.

CGRP may help protect hair follicles from immune system attacks, potentially slowing hair loss.

taVNS reduces vitiligo symptoms in mice.

Melanocytes may trigger the immune response in alopecia areata, affecting hair regrowth.

People with alopecia areata have more Th17 cells and fewer Treg cells, which may be key to the condition's development.

Skin's Regulatory T cells are crucial for maintaining skin health and could be targeted to treat immune-related skin diseases and cancer.

CD3+ T-cell presence is a reliable marker to tell apart alopecia areata from pattern hair loss.

Tissue-resident memory T cells can protect against infections and cancer but may also contribute to autoimmune diseases.

The role of γδT-cells in causing alopecia areata remains unclear.

Regulatory T cells are essential for normal and improved wound healing in mice.

Patients with Alopecia areata have fewer specific immune cells that normally regulate the immune system, which may contribute to the condition.

TNC+ fibroblasts play a key role in skin inflammation by interacting with T cells.

Targeting Vδ1+T-cells may help treat alopecia areata.

Using healthy donor stem cells can potentially calm overactive immune cells and reduce inflammation in severe hair loss patients, offering a possible treatment method.

Malt1 protease is essential for regulatory T cell function and could be targeted to boost antitumor immunity.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

Using fat-derived stem cells with the drug meglumine antimoniate can help control skin disease and reduce parasites in mice with leishmaniasis.

Regulatory T cells help heal skin and grow hair, and their absence can lead to healing issues and hair loss.

Anti-IL-12/IL-23 antibody therapy effectively treats psoriasiform skin lesions in IBD patients.

Using low-dose IL-2 to increase regulatory T cells might be a safe way to treat type 1 diabetes without severe side effects.

Stress increases nerve fibers and immune cell activity in mouse skin, possibly worsening skin conditions.

Some patients with mycosis fungoides or Sézary syndrome experience hair loss, which may be similar to alopecia areata or linked to skin lesions, possibly due to abnormal T cells, and bexarotene can help treat it.

ATIR101 improves survival in stem cell transplant patients; Australian stem cell treatment decisions are influenced by regulation changes.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

The study's results on the effectiveness of low-dose IL-2 for alopecia areata and its impact on immune cells were not provided.