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Mechlorethamine treatment regrew hair in mice by killing immune cells causing hair loss without harming hair follicles.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

Hair loss in mice starts with immune cells damaging hair roots before it becomes visible.

Different types of stem cells in the skin contribute to the variety of melanoma forms.

Tissue environment greatly affects the unique epigenetic makeup of regulatory T cells, which could impact autoimmune disease treatment.

Mice treatments didn't grow hair, a patient treatment may affect immune response, and people with hair loss often feel anxious or depressed.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Keratinocytes help heal skin wounds by interacting with immune cells and producing substances that kill pathogens.

The research found that different types of fibroblasts are involved in wound healing and that some blood cells can turn into fat cells during this process.

Different markers are found in stem cells of the scalp's hair follicle bulge and the surrounding skin.

Tofacitinib was more effective than apremilast in treating hair loss in a mouse model of alopecia areata.

Deleting the BRD4 protein in certain skin cells causes hair loss and skin inflammation.

Alopecia areata involves immune system imbalances that may lead to depression and anxiety.

Lichen planus is a chronic autoimmune disease that is hard to treat and more common in women.

Key genes linked to immune response are upregulated in hair follicles and skin tissues in chronic discoid lupus erythematosus.

Key genes linked to immune response are highly active in lupus-affected hair follicles.

Human hair follicles have a unique way of using energy and might use the Cori cycle; blocking CCR5 could help treat hair loss.

Frontal Fibrosing Alopecia's cause is unclear, affects mainly postmenopausal women, and current treatments focus on stopping hair loss rather than regrowth.

A specific DNA sequence caused hair loss in male mice by activating immune cells and increasing a certain immune signal.

The skin has a complex immune system that is essential for protection and healing, requiring more research for better wound treatment.

Stress can cause hair loss by negatively affecting hair follicles and this effect might be reversed with specific treatments.

Psoriasis is linked to other autoimmune diseases and involves a specific inflammatory process.

Symmetrical Acrokeratoderma (SAK) may be a unique skin condition in China, lacking specific treatment and needing long-term monitoring.

Cells from the lower part of hair follicles are a promising, less invasive option for immune system therapies.

Systemic corticosteroids can help treat rapidly spreading vitiligo but require more research for safer use.

Men generally have more severe COVID-19 cases and higher death rates than women due to biological differences.

Human prenatal skin develops an immune network early on that helps with skin formation and healing without scarring.

The document concludes that alopecia has significant social and psychological effects, leading to a market for hair loss treatments.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

UV light helped human hair transplants survive in mice without broad immunosuppression.